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EFFECTS OF RITUXIMAB ON THE PROGRESSION OF TYPE 1 DIABETES IN NEW ONSET SUBJECTS

EFFECTS OF RITUXIMAB ON THE PROGRESSION OF TYPE 1 DIABETES IN NEW ONSET SUBJECTS
利妥昔单抗对新发受试者 1 型糖尿病进展的影响
批准号：
7951724
负责人：
JOHN A WAGNER
金额：
$0.4万
依托单位：
UNIVERSITY OF MINNESOTA
依托单位国家：
美国
项目类别：
财政年份：
2008
资助国家：
美国
项目状态：
已结题
起止时间：
2008-12-01 至 2009-11-30
来源：
https://reporter.nih.gov/project-details/7951724
关键词：
B-LymphocytesBeta CellClinical ResearchComputer Retrieval of Information on Scientific Projects DatabaseDependenceDiabetes MellitusDiseaseFundingGrantImmuneInstitutionInsulinInsulin-Dependent Diabetes MellitusLifeMediatingResearchResearch PersonnelResourcesSourceUnited States National Institutes of Healthpreventrituximab

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中文摘要
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英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Type 1 diabetes mellitus is an immune-mediated disease in which insulin-producing beta cells are completely destroyed resulting in life-long dependence on exogenous insulin. We hypothesize that rituximab, by eliminating B cells, would be effective at halting beta cell destruction thereby preventing the progression of diabetes.
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CALCIUM DYNAMICS AND EGG ACTIVATION
CALCIUM DYNAMICS AND EGG ACTIVATION
IMPROVING AAV VECTOR TRANSDUCTION IN AIRWAY CELLS
  • 批准号：
    2600702
  • 项目类别：
  • 资助金额：
    $8.39万
  • 财政年份：
    1998
  • 负责人：
    JOHN A WAGNER
  • 依托单位：
    STANFORD UNIVERSITY
MOLECULAR MECHANISMS OF NEUROLOGICAL DISEASE
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