KIDNEY INJURY MOLECULE-1 AND DIABETIC NEPHROPATHY

分子 1 肾损伤和糖尿病肾病

• 批准号: 8359905
• 负责人: Xueying Zhao
• 金额: $ 12.47万
• 依托单位: MOREHOUSE SCHOOL OF MEDICINE
• 依托单位国家: 美国
• 财政年份: 2011
• 资助国家: 美国
• 起止时间: 2011-07-01 至 2012-06-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8359905
• 关键词: Clinical Research; Diabetic Nephropathy; Funding; Goals; Grant; National Center for Research Resources; Pathogenesis; Principal Investigator; Research; Research Infrastructure; Resources; Role; Source; Translational Research; United States National Institutes of Health; cost; rat KIM-1 protein; research study

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. Primary support for the subproject and the subproject's principal investigator may have been provided by other sources, including other NIH sources. The Total Cost listed for the subproject likely represents the estimated amount of Center infrastructure utilized by the subproject, not direct funding provided by the NCRR grant to the subproject or subproject staff. The overall goal of this project remains the same and is to elucidate the regulatory mechanism and functional role of kidney injury molecule-1 (Kim-1) in the pathogenesis of diabetic nephropathy. Experiments performed over the project period have resulted in significant progress on the two Specific Aims.

该子项目是利用资源的众多研究子项目之一 由 NIH/NCRR 资助的中心拨款提供。子项目的主要支持 并且子项目的主要研究者可能是由其他来源提供的， 包括其他 NIH 来源。 子项目可能列出的总成本 代表子项目使用的中心基础设施的估计数量， NCRR 赠款不直接向子项目或子项目工作人员提供资金。 该项目的总体目标保持不变，就是阐明肾损伤分子1（Kim-1）在糖尿病肾病发病机制中的调节机制和功能作用。项目期间进行的实验在两个具体目标方面取得了重大进展。