MOLECULAR DEFECTS OF INSULIN SIGNALING IN PCOS

PCOS 中胰岛素信号传导的分子缺陷

• 批准号: 7606134
• 负责人: Ricardo Azziz
• 金额: $ 0.47万
• 依托单位: LUNDQUIST INSTITUTE FOR BIOMEDICAL INNOVATION AT HARBOR-UCLA MEDICAL CENTER
• 依托单位国家: 美国
• 财政年份: 2007
• 资助国家: 美国
• 起止时间: 2007-02-01 至 2007-11-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7606134
• 关键词: Acute; Adrenal Glands; Androgens; Computer Retrieval of Information on Scientific Projects Database; Corticotropin; Defect; Family; Funding; Grant; Inherited; Institution; Molecular; Patients; Polycystic Ovary Syndrome; Research; Research Personnel; Resources; Source; United States National Institutes of Health; insulin signaling; response

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The study aims to determine whether Adrenal Androgen and Androgen response to acute ACTH stimulation is inherited within families of patients with Polycystic Ovary Syndrome.

这个子项目是许多研究子项目中的一个 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得了主要资金， 因此可以在其他CRISP条目中表示。所列机构为 研究中心，而研究中心不一定是研究者所在的机构。 该研究旨在确定肾上腺雄激素和雄激素对急性ACTH刺激的反应是否在多囊卵巢综合征患者的家族中遗传。