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MOLECULAR MECHANISMS OF NEUTROPHIL MEDIATED TISSUE INJURY IN PERIODONTITIS

MOLECULAR MECHANISMS OF NEUTROPHIL MEDIATED TISSUE INJURY IN PERIODONTITIS
中性粒细胞介导的牙周炎组织损伤的分子机制
批准号：
7606216
负责人：
THOMAS Elliott VAN DYKE
金额：
$7.29万
依托单位：
BOSTON UNIVERSITY MEDICAL CAMPUS
依托单位国家：
美国
项目类别：
财政年份：
2007
资助国家：
美国
项目状态：
已结题
起止时间：
2007-01-03 至 2007-11-30
来源：
https://reporter.nih.gov/project-details/7606216
关键词：
Computer Retrieval of Information on Scientific Projects DatabaseFundingGoalsGrantHumanInflammationInflammatoryInjuryInstitutionMediatingMediator of activation proteinMolecularPeriodontal DiseasesPeriodontitisProductionPurposeResearchResearch PersonnelResourcesSourceTissuesUnited States National Institutes of Healthneutrophilnovel therapeutics

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中文摘要
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英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The purpose of this project is to study the mechanisms that regulate production by human neutrophils of inflammatory mediators that contribute to periodontal disease. Long term goals are to elucidate endogenous counter-regulatory mechanisms in inflammation, and to provide new therapeutic approaches to periodontal diseases.
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