EGFR Activation in H. Pylori-Induced Gastric Cancer

幽门螺杆菌诱发的胃癌中的 EGFR 激活

• 批准号: 7617406
• 负责人: D Brent Polk
• 金额: $ 37.17万
• 依托单位: VANDERBILT UNIVERSITY
• 依托单位国家: 美国
• 财政年份: 2008
• 资助国家: 美国
• 起止时间: 2008-12-01 至 2013-11-30
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7617406
• 关键词: Abbreviations; Address; Animals; Apoptosis; Apoptosis DNA Damage Pathway; Apoptotic; Attenuated; Binding; Biological; Cell Culture Techniques; Cell Line; Cell Survival; Cells; DNA Damage; Data; Development; Disintegrins; Dominant-Negative Mutation; Dysplasia; Epidermal Growth Factor; Epidermal Growth Factor Receptor; Epithelial; Epithelial Cells; Epithelium; Extracellular Signal Regulated Kinases; Family member; Gastrointestinal Diseases; Generations; Genetic; Goals; Helicobacter Infections; Helicobacter pylori; Heparin Binding; Human; Hydrogen Peroxide; In Vitro; Inflammation; Injury; Intestinal Neoplasms; Ligands; Malignant Neoplasms; Matrilysin; Matrix Metalloproteinases; Mediating; Mediator of activation protein; Metalloproteases; Mitogen-Activated Protein Kinases; Modeling; Molecular; Mus; Nature; Oxidative Stress; Pathogenesis; Pathway interactions; Principal Investigator; Program Research Project Grants; Receptor Protein-Tyrosine Kinases; Regulation; Resistance; Role; Signal Transduction Pathway; Stomach; Study models; Text; Transactivation; Tumor Necrosis Factor-alpha; Tumor Necrosis Factors; carcinogenesis; defined contribution; in vivo; malignant stomach neoplasm; mouse model; mutant; novel; oxidative DNA damage; polyamine oxidase; prevent; programs; receptor; repaired; response; transcription factor; tumorigenesis

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