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Elucidating Regulatory Intracellular Mechanisms of Multimeric MAVS Aggregation and Subsequent Pathophysiological Outcomes in Myocardial Infarction

Elucidating Regulatory Intracellular Mechanisms of Multimeric MAVS Aggregation and Subsequent Pathophysiological Outcomes in Myocardial Infarction
阐明心肌梗死中多聚体 MAVS 聚集的细胞内调节机制和随后的病理生理结果
批准号：
486404
负责人：
Haggerty Teagan G
金额：
$1.27万
依托单位：
University of Ottawa
依托单位国家：
加拿大
项目类别：
Studentship Programs
财政年份：
2022
资助国家：
加拿大
项目状态：
已结题
起止时间：
2022-12-01 至 2023-12-01
来源：
https://webapps.cihr-irsc.gc.ca/decisions/p/project_details.html?applId=486404&lang=en
关键词：
Autophagy Cardiac Function Cardiac Remodeling Inflammation And Fibrosis Innate Immunity Mitochondrial Antiviral-Signalling (Mavs) Protein Mitochondrial Dysfunction Mitophagy Myocardial Infarction Ubiquitin-Proteasome Systems

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Mitochondrial Antiviral Signalling (MAVS) is a caspase recruitment domain (CARD)-containing protein, residing uniquely on mitochondrial surfaces and is classically involved in viral innate immune signalling. MAVS has since been recognized for its noncanon
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