Fine particulate matter air pollution and blood pressure: the modifying role of psychosocial stress.
Fine particulate matter air pollution and blood pressure: the modifying role of psychosocial stress.
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DOI：
10.1016/j.envres.2014.06.001
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发表时间：
2014-08
影响因子：
8.3
通讯作者：
Max, Paul
中科院分区：
文献类型：
--
作者：
Hicken, Margaret T.;Dvonch, J. Timothy;Schulz, Amy J.;Mentz, Graciela;Max, Paul
人们日益认识到有必要调查社会心理压力和环境危害对健康的共同影响。一些证据表明，社会心理压力可能是空气污染呼吸系统结果之间关联的重要调节因素，但很少有人研究心血管结果。我们研究了社会心理压力对细颗粒物空气污染 (PM2.5) 和血压 (BP) 之间关联的影响。我们的数据来自底特律健康环境合作伙伴 (HEP) 2002-2003 年调查。在 919 名参与者中，在两个时间点收集了 347 名参与者的血压。基于之前报告该样本中 PM2.5 和血压之间关联的工作，我们在单独的线性模型中对收缩压 (SBP) 和舒张压 (DBP) 以及脉压 (PP) 进行了回归，了解心理社会压力、PM2.5 和 HEP 社区（西南、东区、西北）之间的相互作用。对于那些报告压力水平较高的人来说，PM2.5 和 SBP 之间的关联性更强，但这种相互作用仅在底特律西南部地区显着。报告低压力的底特律西南部居民显示，前 2 天接触 PM2.5 每增加 10μg/m3，SBP 就会增加 2.94 mmHg (95%CI：-0.85，6.72)。那些报告有高压力的人的 PM2.5 暴露量每增加 10μg/m3，SBP 就会增加 9.05 mmHg (95%CI: 3.29, 14.81)。这些结果表明，社会心理压力可能会增加对 PM2.5 高血压影响的脆弱性。这项工作有助于了解社会和自然环境可能共同导致健康状况不佳和健康差异的方式。
Consensus is growing on the need to investigate the joint effects of psychosocial stress and environmental hazards on health. Some evidence suggests that psychosocial stress may be an important modifier of the association between air pollution respiratory outcomes, but few have examined cardiovascular outcomes. We examined the modifying effect of psychosocial stress on the association between fine particulate matter air pollution (PM2.5) and blood pressure (BP). Our data came from the Detroit Healthy Environments Partnership (HEP) 2002–2003 survey. Of 919 participants, BP was collected at two time points for a subset of 347. Building on previous work reporting associations between PM2.5 and BP in this sample, we regressed systolic (SBP) and diastolic (DBP) BP and pulse pressure (PP), in separate linear models, on the interaction among psychosocial stress, PM2.5, and HEP neighborhood (Southwest, Eastside, Northwest). The association between PM2.5 and SBP was stronger for those who reported high levels of stress, but this interaction was significant only in the Southwest Detroit neighborhood. Southwest Detroit residents who reported low stress showed a 2.94 mmHg (95%CI: −0.85, 6.72) increase in SBP for each 10μg/m3 increase in 2-day prior PM2.5 exposure. Those who reported high stress showed a 9.05 mmHg (95%CI: 3.29, 14.81) increase in SBP for each 10μg/m3 increase in PM2.5 exposure. These results suggest that psychosocial stress may increase vulnerability to the hypertensive effects of PM2.5. This work contributes to an understanding of the ways in which the social and physical environments may jointly contribute to poor health and to health disparities.
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影响因子：
10.4
作者：
Clougherty JE;Kubzansky LD
通讯作者：
Kubzansky LD
影响因子：
10.4
作者：
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通讯作者：
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2.1
作者：
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影响因子：
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作者：
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10.4
作者：
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通讯作者：
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