Understanding eating in the absence of hunger among young children: a systematic review of existing studies.
Understanding eating in the absence of hunger among young children: a systematic review of existing studies.
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DOI：
10.1016/j.appet.2014.10.032
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发表时间：
2015-02
期刊：
影响因子：
5.4
通讯作者：
Gilbert-Diamond, Diane
中科院分区：
文献类型：
--
作者：
Lansigan, Reina K.;Emond, Jennifer A.;Gilbert-Diamond, Diane
在不饿的情况下进食（EAH），或者在饱腹时食用非常美味的食物，是可能导致儿童肥胖的一种行为途径。EAH是一种客观的、基于实验室的测量方法。更全面地了解EAH的潜在决定因素可以为实验室之外的儿童肥胖项目提供信息。对EAH实验进行系统回顾，以确定12岁或以下儿童EAH的个体、家族和社会水平相关因素。从5个电子数据库（Medline [PubMed]、Web of Science、Cochrane Library、CINAHL、PsycINFO）中检索了1487项研究。符合条件的研究是那些最初在实验室环境中测量EAH的研究，招募≤12岁的儿童，或报告≤12岁儿童的特定年龄结果。只包括用英语写的文章。本综述包括12项横断面研究、6项前瞻性研究和1项行为干预研究。EAH在男孩和女孩中均可观察到；EAH的绝对水平随着年龄的增长而增加；母亲的喂养方式与女孩的EAH有关。最一致的证据支持超重和肥胖儿童与正常体重儿童相比EAH水平增加，无论是横断面还是前瞻性。两项研究支持EAH的遗传成分。纳入独立样本的研究支持儿童体重状况与EAH之间的正相关；需要对因果关系进行研究。其他各种个体、遗传和熟悉的特征与EAH相关，但需要在更异质的样本群体中进行研究来证实这些发现。缺少与EAH相关的社会层面因素的研究。
Eating in the Absence of Hunger (EAH), or consuming highly palatable foods when satiated, is one behavioral pathway that may lead to childhood obesity. EAH is an objective, laboratory-based measure. A more comprehensive understanding of potential determinants of EAH could inform childhood obesity programs outside of a laboratory setting. Systematic review of EAH experiments to identify individual, familial, and societal-level correlates of EAH among children 12 years of age or younger. 1,487 studies were retrieved from five electronic databases (Medline [PubMed], Web of Science, Cochrane Library, CINAHL, PsycINFO). Eligible studies were those that measured EAH as initially operationalized in a laboratory setting enrolling children ≤12 years or reporting age-specific results for children ≤12 years. Only articles written in English were included. 12 cross-sectional, six prospective, and one behavioral-intervention studies were included in the review. EAH was observable among boys and girls; absolute levels of EAH increased with age; and maternal feeding styles were associated with EAH among girls. The most consistent evidence supported increased levels of EAH among overweight and obese versus normal weight children, both cross-sectionally and prospectively. Two studies supported a genetic component to EAH. Studies enrolling independent samples support a positive association between weight status and EAH among children; studies addressing causality are needed. Other various individual, genetic, and familiar characteristics were associated with EAH, yet studies among more heterogeneous sample populations are needed to confirm findings. Studies addressing societal-level factors related to EAH were absent.
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