Identification of endogenous retroviral sequences as potential donors for recombinational repair of mutant retroviruses: positions of crossover points.

Identification of endogenous retroviral sequences as potential donors for recombinational repair of mutant retroviruses: positions of crossover points.
复制标题

鉴定内源逆转录病毒序列作为突变逆转录病毒重组修复的潜在供体:交叉点的位置。

DOI:
10.1016/0042-6822(87)90030-4
复制
发表时间:
1987
期刊:
影响因子:
3.7
通讯作者:
Goff,SP
Goff,SP
中科院分区:
医学3区
文献类型:
--
作者:
Colicelli,J;Goff,SP

文献摘要

参考文献

被引文献

相似文献

在基因组重要区域中携带缺失的莫洛尼鼠白血病病毒突变体在感染小鼠细胞后可以通过与内源逆转录病毒序列重组而恢复。我们已经鉴定出含有两个此类重组事件的潜在供体序列的克隆DNA,并确定了相关区域的核苷酸序列。将这些序列与原始突变体和回复病毒的序列进行比较,可以确定用于形成复活体的交叉点。每次交换都发生在两个亲本序列之间具有完美同源性的短片段(17-24 bp)中。
Mutants of Moloney murine leukemia virus carrying deletions in essential regions of the genome can revert after infection of mouse cells by recombination with endogenous retroviral sequences. We have identified cloned DNAs containing potential donor sequences for two such recombination events and determined the nucleotide sequences in the relevant regions. Comparison of these sequences with that of the original mutants and the revertant viruses allowed a determination of the crossover points that were used in formation of the revenants. Each crossover occurred in short stretches (17–24 bp) of perfect homology between the two parent sequences.
正常小鼠细胞中存在的小鼠白血病病毒特异性 DNA 的表征。
DOI: 10.1038/newbio244076a0
发表时间: 1973
期刊: Nature: New biology
影响因子: --
作者:
Lawrence D. Gelb;Julie B. Milstien;Malcolm A. Martin;S. Aaronson
通讯作者: S. Aaronson
小鼠基因组中内源性异种鼠白血病病毒原病毒 DNA 的组织和稳定性
DOI: --
发表时间: 1983
影响因子: 5.4
作者:
M. Hoggan;C. Buckler;J. Sears;W. Rowe;M. Martin
通讯作者: M. Martin
DOI: 10.1016/0092-8674(82)90011-3
发表时间: 1982-01-01
期刊: CELL
影响因子: 64.5
作者:
JUNGHANS, RP;BOONE, LR;SKALKA, AM
通讯作者: SKALKA, AM
DOI: 10.1016/0092-8674(80)90629-7
发表时间: 1980-01-01
期刊: CELL
影响因子: 64.5
作者:
KESHET, E;SHAUL, Y;AVIV, H
通讯作者: AVIV, H
C3H/HeN 小鼠 DNA 中小鼠白血病病毒相关 DNA 序列的分子克隆和表征
DOI: 10.1128/jvi.43.1.113-126.1982
发表时间: 1982
影响因子: 5.4
作者:
R. Roblin;Janet Young;R. Mural;Thomas E. Bell;N. James;Ihle
通讯作者: Ihle