亜鉛欠乏土壌におけるイネの生育を向上させる候補遺伝子の特定

随后对不同耐缺锌品种的树冠组织进行了转录组分析。在这项研究中，她通过对几种锌高效和锌低效基因的两组对比组的RNA测序，强调了早期锌缺乏在树冠组织中触发的分子机制。这一方法使她能够1)确定几个新的和众所周知的锌转运蛋白，这些转运蛋白参与锌从树冠到茎和根的转运，以响应缺锌；2)确定缺锌触发可溶性糖到淀粉的转化；3)检测几个可能影响锌效率的候选基因，包括单糖转运蛋白、含锌指结构域的蛋白、赤霉素刺激的家族蛋白和质膜多肽家族蛋白。这项研究已经提交并正在《实验植物学杂志》上进行审查：

Studies were followed by a transcriptome analysis of crown tissue of genotypes contrasting for Zn deficiency tolerance. In this study she highlighted the molecular mechanisms triggered by early Zn deficiency in crown tissue, through RNA sequencing of two contrasting groups of several Zn efficient and Zn inefficient genotypes. This method allowed her to 1) identify several novel and well-known Zn transporters involved in Zn retranslocation from the crown to the shoot and roots in response to Zn deficiency; 2) determine that Zn deficiency triggers the conversion of soluble sugars into starch; and 3) detect several candidate genes possibly conferring Zn efficiency, including a monosaccharide transporter, a Zn finger domain-containing protein, a gibberellin stimulated family protein and a plasma membrane polypeptide family protein. This work has been submitted and is under review in the Journal of Experimental Botany: