亜鉛欠乏土壌におけるイネの生育を向上させる候補遺伝子の特定

研究随后进行了转录组分析的冠组织的基因型对比缺锌耐受性。在这项研究中，她强调了冠组织中早期锌缺乏引发的分子机制，通过对两组对比的几种锌高效和锌低效基因型进行RNA测序。这种方法使她能够1）鉴定出几种新的和众所周知的锌转运蛋白，这些转运蛋白参与锌从冠部到地上部和根部的再转运，以响应锌缺乏; 2）确定锌缺乏引发可溶性糖转化为淀粉;和3）检测几个可能赋予锌效率的候选基因，包括单糖转运蛋白，含锌指结构域的蛋白，赤霉素刺激家族蛋白和质膜多肽家族蛋白。这项工作已提交给《实验植物学杂志》，并正在审查中：

Studies were followed by a transcriptome analysis of crown tissue of genotypes contrasting for Zn deficiency tolerance. In this study she highlighted the molecular mechanisms triggered by early Zn deficiency in crown tissue, through RNA sequencing of two contrasting groups of several Zn efficient and Zn inefficient genotypes. This method allowed her to 1) identify several novel and well-known Zn transporters involved in Zn retranslocation from the crown to the shoot and roots in response to Zn deficiency; 2) determine that Zn deficiency triggers the conversion of soluble sugars into starch; and 3) detect several candidate genes possibly conferring Zn efficiency, including a monosaccharide transporter, a Zn finger domain-containing protein, a gibberellin stimulated family protein and a plasma membrane polypeptide family protein. This work has been submitted and is under review in the Journal of Experimental Botany: