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中文摘要
翻译
摘要 行政核心的目标是整合所有的科学和行政职能, 癌症系统生物学联盟研究中心(U54)。行政核心将是主要的 项目主任和其他主要和关键研究人员组织和开展 这是一个多机构互动研究中心。核心将催化和刺激研究, 外联举措,并作为所有调查员的主要行政资源,提供服务, 支持所有研究项目和核心。这将包括协调小组成员之间的活动, 管理预算,安排调查员和咨询委员会会议,监测 维持对人类研究方案的批准。行政核心将确保里程碑 和最后期限得到满足,并在此过程中,培养一个统一的合议气氛,促进科学 生产力该核心的一项核心和高度优先活动是提供基础设施和监督所有 中心的努力、资源和成果。核心小组还将管理中心内部试点项目 程序.最后,行政核心将直接与CSBC项目主任和其他CSBC 研究中心安排定期电话会议并组织年度研究者会议。
英文摘要
ABSTRACT The objective of the Administrative Core is to integrate all of the scientific and administrative functions of the Research Center for Cancer Systems Biology Consortium (U54). The Administrative Core will be the primary means by which the Program Director and other principle and key investigators organize and carry out the functions of this interactive multi-institutional research center. The core will catalyze and stimulate research and outreach initiatives, and serve as the major administrative resource for all investigators, providing service and support for all research projects and cores. This will include coordinating activities among group members, management of the budget, scheduling of investigator and advisory board meetings, monitoring the status and maintaining the approval of protocols for human research. The Administrative Core will ensure that milestones and deadlines are met and, in the process, foster a unified collegial atmosphere that facilitates scientific productivity. A central and high priority activity of this Core is to provide infrastructure and oversight of the all efforts, resources, and deliverables of the center. The Core will also administer the intra-center pilot project program. Lastly, the Administrative Core will work directly with the CSBC Program Director and other CSBC Research Centers to schedule regular conference calls and organize the annual investigators' meeting.
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AKT as a resistance mechanism to cell cycle and endocrine therapies in ER+ breast cancer
Mechanism of estrogen independent proliferation in ER+ breast cancer cells
Mechanism of estrogen independent proliferation in ER+ breast cancer cells
Evolution of cancer cell phylogenies and phenotypes in breast cancer resistance
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