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DISUSE MUSCLE ATROPHY--MECHANISM OF DEVELOPMENT

DISUSE MUSCLE ATROPHY--MECHANISM OF DEVELOPMENT
废用性肌萎缩——发展机制
批准号：
6115159
负责人：
CHARLES A STUART
金额：
$3.42万
依托单位：
UNIVERSITY OF TEXAS MED BR GALVESTON
依托单位国家：
美国
项目类别：
财政年份：
1998
资助国家：
美国
项目状态：
已结题
起止时间：
1998-12-01 至 1999-11-30
来源：
https://reporter.nih.gov/project-details/6115159
关键词：
atrophy clinical research gene expression human subject insulinlike growth factor messenger RNA muscle metabolism protein biosynthesis rest restricted physical activity striated muscles

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中文摘要
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英文摘要
Tests the hypothesis that a direct consequence of muscular inactivity in man is decreased skeletal muscle protein synthesis and that activity related modulation of protein synthesis is mediated via changes in local production of insulin-like growth factor I (IGF-I). The protein synthetic rate in skeletal muscle will be directly measured in normal volunteers subjected to absolute bedrest for seven days. Molecular biology techniques will be employed to quantitate alterations in skeletal muscle messenger RNA and IGF-I.
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  • 依托单位：
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  • 依托单位：
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Mechanisms by which exercise training ameliorates the metabolic syndrome
  • 批准号：
    8035605
  • 项目类别：
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INSULIN RESISTANCE, POLYCYSTIC OVARIAN DISEASE AND ACANTHOSIS NIGRICANS
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