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Mechanisms of Arterial Dysfunction in Type 2 Diabetes Mellitus

Mechanisms of Arterial Dysfunction in Type 2 Diabetes Mellitus
2 型糖尿病动脉功能障碍的机制
批准号：
7524091
负责人：
MARK A CREAGER
金额：
$33.23万
依托单位：
BRIGHAM AND WOMEN'S HOSPITAL
依托单位国家：
美国
项目类别：
财政年份：
2007
资助国家：
美国
项目状态：
已结题
起止时间：
2007-04-01 至 2010-03-31
来源：
https://reporter.nih.gov/project-details/7524091
关键词：
1-Phosphatidylinositol 3-KinaseAccountingAdipocytesAdipose tissueAgreementAmputationAntiatherogenicAtherosclerosisAttenuatedBiological AssayBiological PreservationBiologyBiopsy SpecimenBlood VesselsC-reactive proteinCD40 LigandCardiovascular systemCause of DeathCerebrovascular DisordersClinicalCoronary ArteriosclerosisCross-Over StudiesCrossover DesignCutaneousDeoxyglucoseDevelopmentDiabetes MellitusDiffuseDoseEndotheliumEuglycemic ClampingEventExperimental ModelsFat emulsionForearmFunctional disorderGlucoseGlucose ClampGoalsHeat-Shock Proteins 90HeparinHomeostasisHyperglycemiaInflammationInflammation MediatorsInflammatoryInfusion proceduresInsulinInsulin ReceptorInsulin ResistanceInterleukin-6InvestigationLeadMeasurementMeasuresMediatingMediator of activation proteinMolecularMuscleMyocardial InfarctionNF-kappa BNitric OxideNon-Insulin-Dependent Diabetes MellitusNonesterified Fatty AcidsPathway interactionsPatientsPeripheralPeripheral arterial diseasePharmaceutical PreparationsPhosphorylationPhosphotransferasesPlacebo ControlPlacebosPlasmaPlethysmographyPositron-Emission TomographyPrevalencePrincipal InvestigatorProductionProgram Research Project GrantsProtein Kinase CProto-Oncogene Proteins c-aktRandomizedRegulationResearch PersonnelResistanceResolutionRho-associated kinaseRiskSerineSeveritiesSignal PathwaySignal TransductionSkeletal MuscleSkeletal systemSkinStrokeTestingTherapeuticTissuesTumor Necrosis Factor-alphaTumor Necrosis FactorsTyrosine PhosphorylationUltrasonographyVascular DiseasesVasodilationVenousabstractingacipimoxbrachial arterycytokinediabeticdisabilityfasudilfatty acid metabolismglucose uptakehuman NOS3 proteinhuman TNF proteinhuman subjectimprovedin vivoinsightinsulin receptor substrate 1 proteininsulin secretioninsulin sensitivityinsulin signalingkinase inhibitormuscle metabolismprogramsresearch studysalicylsalicylic acidtherapeutic targettreatment duration

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