MECHANISMS OF NO-CONTRIBUTION TO REFLEX CUTANEOUS VASODILATION WITH HYPERTENSIO

这个子项目是许多研究子项目中利用 资源由NIH/NCRR资助的中心拨款提供。子项目和 调查员(PI)可能从NIH的另一个来源获得了主要资金， 并因此可以在其他清晰的条目中表示。列出的机构是 该中心不一定是调查人员的机构。 本研究的目的是确定精氨酸酶在健康老年受试者(65-85岁)和健康年轻受试者(18-30岁)反射性皮肤血管扩张中的作用，并与健康年轻受试者组比较，检测活性氧在非依赖部分反射性皮肤血管扩张中的相对作用。

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The purpose of the study is to determine the role of arginase in reflex cutaneous vasodilation in a healthy aged human subject group (65-85 years old) compared to a healthy young subject group (18-30 years old) and to examine the relative role of reactive oxygen species on the NO-dependent portion of reflex cutaneous vasodilation in an aged humans subject group compared to a healthy young subject group.