TULANE COBRE: OXYGENASE-CARBON MONOXIDE SYSTEM IN SALT-INDUCED HYPERTENSION

这个子项目是许多研究子项目中利用 资源由NIH/NCRR资助的中心拨款提供。子项目和 调查员(PI)可能从NIH的另一个来源获得了主要资金， 并因此可以在其他清晰的条目中表示。列出的机构是 该中心不一定是调查人员的机构。 在项目3中，Johnson评估了这样的假设，即血管中的血红素加氧酶(HO)形成一氧化碳(CO)，该一氧化碳与一氧化氮合酶(NOS)的血红素部分结合，从而减少NO的形成。

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. In Project 3, Johnson evaluates the hypothesis that vascular heme oxygenase (HO) forms carbon monoxide (CO) that binds to the heme moiety of nitric oxide (NO) synthase (NOS) and decreases the formation of NO.