TULANE COBRE: OXYGENASE-CARBON MONOXIDE SYSTEM IN SALT-INDUCED HYPERTENSION

该子项目是利用NIH/NCRR资助的中心赠款提供的资源的许多研究子项目之一。子项目和研究者（PI）可能从另一个NIH来源获得了主要资金，因此可以在其他CRISP条目中表示。所列机构为中心，不一定是研究者所在机构。在项目3中，约翰逊评估了血管血红素加氧酶（HO）形成一氧化碳（CO）的假设，该一氧化碳与一氧化氮（NO）合酶（NOS）的血红素部分结合并减少NO的形成。

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. In Project 3, Johnson evaluates the hypothesis that vascular heme oxygenase (HO) forms carbon monoxide (CO) that binds to the heme moiety of nitric oxide (NO) synthase (NOS) and decreases the formation of NO.