课题基金 / 基金详情

UCLA-Caltech Medical Scientist Training Program

UCLA-Caltech Medical Scientist Training Program
加州大学洛杉矶分校-加州理工学院医学科学家培训计划
批准号:
10632114
负责人:
Olujimi A Ajijola
金额:
$145.49万
依托单位国家:
美国
项目类别:
财政年份:
1983
资助国家:
美国
项目状态:
已结题
起止时间:
1983-07-01 至 2024-06-30

项目摘要

项目成果

Olujimi A Ajijola的其他基金

相似基金

相关文献

中文摘要
翻译
点击翻译按钮获取中文摘要
英文摘要
ABSTRACT The mission of the UCLA-Caltech MSTP is to educate and train outstanding physician-scientists. To fulfill this mission, our current goals are to 1) recruit exceptionally bright and accomplished students who exhibit unusual passion for scientific knowledge and a life-long commitment to research, medicine, service, and leadership; 2) help guide admitted students toward outstanding training environments that encourage individual thinking and provide tools for development into accomplished physician-scientists; 3) provide a comprehensive support system to meet trainees' needs; and 4) enhance physician-scientist diversity by playing an increasingly prominent role in guiding the career development of current and future students from under-represented ethnic groups and disadvantaged backgrounds through mentoring and outreach. To accomplish these major goals as effectively as possible, the UCLA-Caltech MSTP is led by two equal Co-Directors, four Associate Directors, and a strong administrative team, all of whom are deeply committed to the Program. The Program is structured for an average of eight years of combined research and clinical training. Our integrated problem-based medical school curriculum emphasizes flexibility and is particularly well suited for MSTP students because it provides increased time for independent scientific exploration and encourages trainees to pursue research projects that will advance current knowledge of disease etiology, diagnosis, and treatment. For Ph.D. training, students choose mentors affiliated with a broad array of UCLA and Caltech graduate programs, most frequently in genetics & genomics, immunology, cell & developmental biology, neuroscience, bioinformatics, bioengineering, or our newly instituted social sciences track. A recent milestone achievement of our Program was the recent 20th anniversary of the affiliation between UCLA and Caltech into a joint MSTP (1997) for the mutual benefit of these preeminent campuses and our trainees. A second remarkable celebratory achievement will be the upcoming centennial of UCLA becoming a UC campus (1919) and its amazing rise to a world-ranked institution in research, education, and service in under 100 years of existence. A prominent goal of UCLA’s Centennial Campaign is to raise $4 billion, one-third of which is earmarked for research. Despite a competitive funding climate, the UCLA- Caltech MSTP is in a period of substantial growth because of generous institutional support from the David Geffen School of Medicine at UCLA and from Caltech. This has permitted an increase in the size of the Program, with 107 current students. The MSTP benefits tremendously from substantial improvements and expansion in physical facilities and research capabilities at UCLA and Caltech, from the strong financial health of both universities, and from the recruitment of many outstanding new faculty members to our campuses.
期刊论文(134)
专著(0)
科研奖励(0)
会议论文
DOI: 10.1016/j.jmb.2015.05.023
发表时间: 2015-07-31
期刊: Journal of molecular biology
影响因子: 5.6
作者: [Owens GE, New DM, West AP Jr, Bjorkman PJ]
通讯作者: Bjorkman PJ
DOI: 10.1021/acs.orglett.9b03530
发表时间: 2019-11-15
期刊: Organic letters
影响因子: 5.2
作者: [Sercel ZP, Sun AW, Stoltz BM]
通讯作者: Stoltz BM
DOI: 10.1007/s11060-023-04531-z
发表时间: 2024-01
期刊: JOURNAL OF NEURO-ONCOLOGY
影响因子: 3.9
作者: [Rinonos, Serendipity Zapanta, Li, Tie, Pianka, Sean Thomas, Prins, Terry J., Eldred, Blaine S. C., Kevan, Bryan M., Liau, Linda M., Nghiemphu, Phioanh Leia, Cloughesy, Timothy F., Lai, Albert]
通讯作者: Lai, Albert
Transforming growth factor beta 1 augments mitogen-induced prostaglandin synthesis and expression of the TIS10/prostaglandin synthase 2 gene both in Swiss 3T3 cells and in murine embryo fibroblasts.
转化生长因子 beta 1 可增强 Swiss 3T3 细胞和小鼠胚胎成纤维细胞中丝裂原诱导的前列腺素合成和 TIS10/前列腺素合酶 2 基因的表达。
DOI: 10.1002/jcp.1041590110
发表时间: 1994
期刊: Journal of cellular physiology
影响因子: 5.6
作者: [Gilbert,RS, Reddy,ST, Kujubu,DA, Xie,W, Luner,S, Herschman,HR]
通讯作者: Herschman,HR
73
    Antiarrhythmic mechanisms of chronic vagal nerve stimulation in sympathetic neurons
    Antiarrhythmic mechanisms of chronic vagal nerve stimulation in sympathetic neurons
    Core A: Ultrastructural Assessment & Human Tissue
    Satellite Glial Cell Activation and Sympathetic Imbalance in Cardiomyopathy and Arrhythmias
    海外基金