The relationship between ocular temperature and ocular injury by environmental temperature and non-ionizing radiation

紫外线（UV）或可见光照射过程中眼内温度升高的模拟Maxwell视图照明或类似聚焦光束情况下人眼中产生的热传递使用数学模型进行模拟，以确定眼睛中诱导的温度升高。结果表明，麦克斯韦视图照明或类似的聚焦光束的情况下，可以造成热损伤的透镜在某些条件下（10 mW照射使透镜温度升高7℃）。白内障形成与红外辐射的关系（700-1,400 nm）、YAG激光（1,064 nm，伊拉）、CO_2激光（10,600 nm，IRC）等。宽带光暴露导致结膜水肿和眼睑肿胀。由于在IR暴露期间这些类型的眼外炎症引起的眼睑闭合，IR没有渗透到眼睛的后部。分别测量伊拉或IRC照射时眼眶、玻璃体、透镜、前房及眼睑的温度。暴露后即刻眼睑温度为50-65℃，前房、玻璃体和眼球后分别为40±1.0℃、38.6±0.1℃、38.2±0.1℃。IRC暴露开始后，前房和后房温度迅速升高至41-42℃，玻璃体温度逐渐升高，但不超过40℃。在IRC exposation.ConclusionsEye损伤后观察到的非电离暴露可能发生通过热效应，由于吸收某些波长，而不是通过非热效应。

Simulation of intraocular temperature rise during exposure of ultraviolet (UV) or visual lightThe transfer of heat generated in the human eye in Maxwellian-view illumination or similar focal-beam situations was simulated using a mathematical model to determine the temperature elevations induced in the eye. It was shown that Maxwellian-view illumination or similar focal-beam situations can cause thermal injury to the lens under certain conditions (The lens temperature increased 7℃ by 10mW exposure).The relationship between cataract formation and infrared exposureOcular injuries by broad band light exposure (700-1,400 nm), YAG laser (1,064 nm、IRA), CO_2 laser (10,600 nm, IRC) were investigated. Broad band light exposure induced conjunctival edema and swollen eyelids. Due to the eyelid closure caused by these types of extraocular inflammation during IR exposure, IR did not penetrate into the posterior part of the eye. YAG laser exposure induced corneal changes and cataract CO_2 exposures caused sever corneal injury, but did not induc cataract.Intraocular temperature changes by IRA or IRC exposureTemperatures of the retrobulbar cavity of the orbit, vitreous, lens, anterior chamber and eyelids were measured during IRA or IRC exposure. The eyelid temperature showed 50-65℃ immediately after exposure, and anterior chamber, vitreous, and retrobullbar were 40±1.0℃, 38.6±0.1℃, 38.2±0.1℃, respectively. The temperature of the anterior and posterior chamber quickly increased to 41-42℃ after the start of IRC exposure.The vitreous temperature gradually increased, but did not exceed 40℃. The retrobluber temperature was almost constant during IRC exposure.ConclusionsEye injuries observed after the non-ionizing exposure might have occurred through the thermal effect due to absorption of certain wavelength, not through the non-thermal effect.