A comprehensive study on the sudden infant death syndrome (SIDS) and lipid metabolism.

为了阐明婴儿猝死综合征（SIDS）的基本机制，我们从生物化学和流行病学的角度检查了几项研究：i）使用肠细胞培养模型通过肉毒碱分子结构的差异评价肉毒碱的肠吸收，ii）肉毒碱在小鼠胶质母细胞瘤中的衰老和抗衰老作用的机制，（三）日本科比炎热夏季日最高温度和个人暴露温度的比较研究。

To clarify the basic mechanism of sudden infant death syndrome (SIDS), we examined several studies from the standpoint of both biochemistry and epidemiology : i) Evaluation of intestinal absorption of carnitine by the difference of its molecular structure using intestinal cell culture model, ii) Mechanism of senescence and anti-aging effect of carnitine in mouse glioblastoma, iii) A comparative study of daily maximum and personally exposed temperatures during hot summer days in Kobe, Japan.