DRUG USE--GENERATIONAL TRANSMISSION IN MINORITY YOUTH

这个项目的目的是研究三种病毒的传播。 几代人都是吸毒/吸毒的童年先驱。这 纵向研究始于1990年(T1)，第一代(G1)母亲 对第二代(G2)青少年进行了评估。数据是 获得关于文化和家庭背景、父母养育子女的信息，以及 个性/行为变量。五年后(T2)，G2 对青少年进行了重新评估。在T2数据收集处，原始的 重复T1测量，并从两个测量中收集附加数据 母亲和孩子对文化的影响，个性/行为，父母- 儿童关系、同伴吸毒和自我吸毒。T1-T2数据 确定青少年因素确实会影响以后的药物使用；我们现在 建议通过研究G3进一步调查这些影响 G2原始研究青少年的后代。G3的孩子将是 在7-12岁时评估儿童后期药物的前体 吸毒/吸毒。然后我们将追踪代内和代际 决定药物倾向特征的途径(T1-T3) 已发送。以我们的家庭为基础的虚拟路径 互动模式，即文化因素与父母人格 风险特征(例如，非常规、情绪控制不良) 导致亲子之间的相互疏离(例如，少一些温暖，多一些 冲突)。这随后会导致儿童患上 吸毒/吸毒。我们假设，这条路径，已经显示 在G1父代和G2子代中，将对G2子代(现在)重复 父母)和他/她的后代。同样有趣的是我们的检查 风险(有利于毒品)和保护性(不利于毒品)的内部和 影响G3儿童结局的代际互动。这个 这项研究的样本大小为325名7-12岁的儿童及其父母。T3 数据将通过自我报告的方式收集。分析技术将会 包括结构方程和层次回归。这个 这项研究的意义在于它划定了代际关系 影响儿童毒品前体发育的因素 吸毒/吸毒。(通过专注于改善心理社会的方法 环境中的幼儿处于危险之中，不仅不能轻 以后使用药物的可能性，但也会影响到改变 代际风险传递的链条。)

The purpose of this project is to study the transmission across three generations of the childhood precursors of drug use/drug abuse. This longitudinal study began in 1990 (T1) when first generation (G1) mothers and second generation (G2) adolescents were assessed. Data were obtained on cultural and family context, parent child-rearing, and personality/behavioral variables. Five years later (T2), the G2 adolescents were reassessed. At the T2 data collection, the original T1 measures were repeated, and additional data were gathered from both mother and child on cultural influences, personality/behavior, parent- child relations, peer drug use and self drug use. The T1-T2 data established that adolescent factors do influence later drug use; we now propose to further investigate these influences by studying the G3 offspring of the G2 original study adolescents. The G3 child will be evaluated at age 7-12 in terms of childhood precursors of later drug use/drug abuse. We will then trace the intra- and intergenerational pathways (T1-T3) to determine how drug-prone characteristics are transmitted. The hypothesized pathway, based on our Family Interactional Model, is that cultural factors and parent personality risk characteristics (e.g. unconventionality, poor control of emotions) lead to parent-child mutual detachment (e.g., less warmth, more conflict). This then leads to the development in the child of risks for drug use/drug abuse. We hypothesize that this pathway, already shown in the G1 parent and G2 child, will be repeated for the G2 child (now a parent) and his/her offspring. Also of interest is our examination of risk (drug-conducive) and protective (nondrug-conducive) intra- and intergenerational interactions as they affect G3 child outcome. The sample size for the study is 325 7-12-year-olds and their parents. T3 data will be collected using self-report. The analytic techniques will include structural equation and hierarchical regression. The significance of the study lies in its delineation of generational factors implicated in the development of childhood precursors of drug use/drug abuse. (By focusing on ways to improve the psychosocial environment of the young child at risk, one can not only lesser the likelihood of later drug use, but also effect changes that "break the chain" of generational risk transmission.)