Molecular analysis of activation mechanism of volume-sensitive chloride channel
Molecular analysis of activation mechanism of volume-sensitive chloride channel
批准号:
19590219
负责人:
TAKAHASHI Nobuyuki
金额:
$2.91万
依托单位国家:
日本
项目类别:
Grant-in-Aid for Scientific Research (C)
财政年份:
2007
资助国家:
日本
项目状态:
已结题
起止时间:
2007 至 2008
中文摘要
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英文摘要
生理的な細胞機能の発現に必須である細胞容積制御機構に関与する容積感受性クロライドチャネル(VSOR)の活性化メカニズムを明らかにする目的で、VSORの活性を制御する可能性が示唆されているKCNE1、ならびにアポトーシス刺激により活性化されるタンパク質リン酸化酵素の細胞容積制御機構における関与を検討した.その結果、いずれのタンパク質もVSOR活性化には関与しなかったものの、タンパク質リン酸化酵素であるJNKとp38がVSOR活性化の下流に位置する細胞内情報伝達系であることが初めて明らかとなった.
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Signaling events employed in the hypertonic activation of cation channels in HeLa cells
HeLa 细胞阳离子通道高渗激活中使用的信号事件
DOI:
--
发表时间:
2007
期刊:
Cell. Physiol. Biochem 20
影响因子:
--
作者:
[Wehner, F., Numata, T., Subramanyam, M., Takahashi, N. and Okada, Y]
通讯作者:
Y
Mechanism of RVI inhibition by apoptotic stimulations
细胞凋亡刺激抑制 RVI 的机制
DOI:
--
发表时间:
2007
期刊:
影响因子:
--
作者:
[Takahashi, N., Subramanian, M., Okada, Y]
通讯作者:
Y
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依托单位:
海外基金