Study for the investigation of etiology of polycystic ovary syndrome (PCOS) using androgen-treated female to male transsexuals as a PCOS model
使用雄激素治疗的女变男变性者作为 PCOS 模型调查多囊卵巢综合征 (PCOS) 病因学的研究
基本信息
- 批准号:20591922
- 负责人:
- 金额:$ 3万
- 依托单位:
- 依托单位国家:日本
- 项目类别:Grant-in-Aid for Scientific Research (C)
- 财政年份:2008
- 资助国家:日本
- 起止时间:2008 至 2010
- 项目状态:已结题
- 来源:
- 关键词:
项目摘要
Androgen exposure mechanism is one of major candidates for the etiology of polycystic ovary syndrome (PCOS). We examined the effects of androgen treatment on female to male transsexuals as a model of PCOS. We got the results that androgen induced secondary PCOS endocrinologically and histologically, even partially. It is suspected that androgen exposure is one of etiology for PCOS.
雄激素暴露机制是多囊卵巢综合征(PCOS)的主要病因之一。我们研究了雄激素治疗对作为PCOS模型的女性变男性的影响。结果表明,雄激素在内分泌和组织学上均可诱发继发性PCOS,甚至部分诱发。雄激素暴露可能是PCOS的病因之一。
项目成果
期刊论文数量(0)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)
Spontaneous ovarian hyperstimulation syndrome and pituitary adenoma : incidental pregnancy triggers a catastrophic even Fertil Steril.
自发性卵巢过度刺激综合征和垂体腺瘤:意外怀孕甚至会引发灾难性的生育不育。
- DOI:
- 发表时间:2009
- 期刊:
- 影响因子:0
- 作者:Baba T;Endo T;Kitajima Y;Kamiya H;Moriwaka O;Saito T.
- 通讯作者:Saito T.
The contributions of resistin and adiponectin gene single nucleotide polymorphisms to the genetic risk for polycystic ovary syndrome in a Japanese population
- DOI:10.1080/09513590902972042
- 发表时间:2009-01-01
- 期刊:
- 影响因子:2
- 作者:Baba, Tsuyoshi;Endo, Toshiaki;Saito, Tsuyoshi
- 通讯作者:Saito, Tsuyoshi
Remarkable features of ovarian morphology and reproductive hormone in insulin-resistant Zucker fatty (fa/fa) rats.
胰岛素抵抗 Zucker 脂肪 (fa/fa) 大鼠卵巢形态和生殖激素的显着特征。
- DOI:
- 发表时间:2010
- 期刊:
- 影响因子:0
- 作者:Honnma H;Endo T;et al.
- 通讯作者:et al.
Distinctive features of female-to-male transsexualism and prevalence of gender disorder in Japan.
日本女性变男性易性症的显着特征和性别障碍的流行。
- DOI:
- 发表时间:2011
- 期刊:
- 影响因子:3.5
- 作者:Baba T;Endo T;et al.
- 通讯作者:et al.
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ENDO Toshiaki其他文献
Side Readings "The Residential Buildings to be built in AKITA"(in Japanese)
旁读《秋田县即将建设的住宅楼》(日语)
- DOI:
- 发表时间:
2003 - 期刊:
- 影响因子:0
- 作者:
ENDO Toshiaki;NISHIKAWA Ryoji;et al. - 通讯作者:
et al.
ENDO Toshiaki的其他文献
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{{ truncateString('ENDO Toshiaki', 18)}}的其他基金
The role of neuronal nitric oxide synthase-expressing neurons in the regulation of synaptic plasticity in the visual cortex
神经元一氧化氮合酶表达神经元在视觉皮层突触可塑性调节中的作用
- 批准号:
22700412 - 财政年份:2010
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for Young Scientists (B)
Study for the etiology of polycystic ovary using a rat model
大鼠多囊卵巢病因的研究
- 批准号:
16591676 - 财政年份:2004
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
The differences of mechanisms between normal luteal formation and occurrence of ovarian hyperstimulation syndrome
正常黄体形成与卵巢过度刺激综合征发生机制的差异
- 批准号:
14571574 - 财政年份:2002
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
Study for the etiology and therapy of polycystic ovary syndrome and ovarian hyperstimulation syndrome.
多囊卵巢综合征和卵巢过度刺激综合征的病因及治疗研究。
- 批准号:
11671637 - 财政年份:1999
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
Common hormones and mechanisms of luteal regression, ovulation and follicular atresia.
黄体退化、排卵和卵泡闭锁的常见激素和机制。
- 批准号:
08671909 - 财政年份:1996
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for Scientific Research (C)
LUTEAL REGRESSION AND PROTEINASE,APOPTOSIS,HEAT SHOCK PROTEIN
黄体退化与蛋白酶、细胞凋亡、热休克蛋白
- 批准号:
06671670 - 财政年份:1994
- 资助金额:
$ 3万 - 项目类别:
Grant-in-Aid for General Scientific Research (C)
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