Genetic and physiological studies on seed coat cracking in soybean

大田条件下大豆种皮开裂。鉴定出两个qtl， cr1（分子连锁基团（MLG） D1b和cr2 MLG M）。在近等基因系（NILs）中，cr1与种子大小或成熟度无关。因此，QTL周围的标记可能对育种有用，在不改变种子大小或成熟度的情况下减少开裂症状。相比之下，在MLG C1中鉴定出两个与网状开裂相关的qtl (ncr1和ncr2)，距离约为62 cM。这些结果表明，在田间条件下发生的种皮开裂与网状开裂的遗传控制不同。基因芯片分析显示，耐药基因在耐药基因中高度表达。26856, Gma. 16827, Gma. 173)和高表达基因在敏感的NIL（Gma. 173）中表达。48109和Gma. 49562)。

The seed coat of soybean cracks under field conditions. Two QTLs, cr1(molecular linkage group(MLG) D1b and cr2 MLG M) were identified. The cr1 was not associated with seed size or maturity in near-isogenic lines(NILs). Therefore, markers around the QTL may be useful for breeding to reduce cracking symptoms without altering seed size or maturity. In contrast, two QTLs for net-like cracking(ncr1 and ncr2) were identified in MLG C1 with a distance of about 62 cM. These results suggest that genetic control of seed coat cracking occurring under field conditions was different from net-like cracking. Microarray of the NILs revealed genes highly expressed in the tolerant NIL(Gma. 26856, Gma. 16827, Gma. 173) and genes highly expressed in the sensitive NIL(Gma. 48109 and Gma. 49562).