Proximal Effects of Alcohol on Same-Sex Intimate Partner Violence

同性亲密关系中的伴侣暴力（SS-IPV）是一个严重不足的公共卫生问题。 问题. SS-IPV致病的病因学模型对于干预措施的制定至关重要，必须包括 同性关系特有的因素（例如，性少数压力）;然而，这种模式尚未得到 开发或验证。对此有三个特别值得注意的解释，包括专门使用横截面 研究SS-IPV犯罪的设计（即，无法建立时间效应），没有研究 酒精对SS-IPV犯罪的近端影响，以及缺乏解释参与者和 他们亲密伴侣的交叉社会身份这些限制阻碍了研究的发展， 测试旨在减少SS-IPV的基于理论和文化敏感的干预措施。 拟议项目的科学前提是优先考虑研究所强调的三个观点。 医学（2011年）报告-少数民族压力，社会生态学和交叉性-同时解决 上述缺点。我们的目的是确定（1）性少数压力对SS-IPV的时间效应 （2）近端饮酒是否会改变性少数压力的阈值 SS-IPV犯罪，（3）性少数压力，近端酒精使用， 其他因素促进SS-IPV，以及（4）这些互动和中介效应如何被改变， SS-IPV的个人和夫妇水平的风险和弹性因素的模式。 这些目标将通过利用实验室为基础的实验（研究1）的互补优势来实现。 和纵向每日日记法（研究2），这是建立时间的金标准方法 危险因素与IPV发病的关系。对于每项研究，我们将招募一个独立的样本， 确定为男同性恋或女同性恋并处于IPV高风险的顺性别男性-男性和女性-女性夫妇 （基于IPV的先前历史），来自佐治亚州亚特兰大。在这两项研究中，将在一个 参与者-合作伙伴相互依赖建模框架，该框架将允许有效分析双方合作伙伴的 在个人和夫妇层面，相互交织的身份以及风险和复原力因素。 拟议项目最重要的贡献将是提供第一个全面的病因学 SS-IPV犯罪模型，该模型同时考虑了干扰和基于过程的因素， 性少数压力和近端酒精效应在个人和夫妇水平的分析。在这样做时， 从这个项目中得出的结果将对发展所需的证据基础做出重大贡献 有效的、了解文化的SS-IPV治疗和预防工作。

Partner violence within same-sex intimate relationships (SS-IPV) is a vastly understudied public health problem. Etiological models of SS-IPV perpetration are critical to intervention development and must include factors unique to same-sex relationships (e.g., sexual minority stress); however, such models have yet to be developed or validated. Three particularly notable explanations for this include exclusive use of cross-sectional designs to study SS-IPV perpetration (i.e., inability to establish temporal effects), no studies which examine the proximal effects of alcohol on SS-IPV perpetration, and a paucity of studies which account for participants’ and their intimate partners’ intersecting social identities. These limitations prevent research from developing and testing theoretically-based and culturally-sensitive interventions designed to reduce SS-IPV. The scientific premise of the proposed project is to prioritize three perspectives highlighted by the Institute of Medicine (2011) report – minority stress, social-ecology, and intersectionality – while addressing the aforementioned weaknesses. We aim to determine (1) the temporal effect of sexual minority stress on SS-IPV perpetration, (2) whether proximal alcohol use alters the threshold at which sexual minority stress contributes to SS-IPV perpetration, (3) the temporal sequence by which sexual minority stress, proximal alcohol use, and other factors facilitate SS-IPV, and (4) how these interactive and mediational effects are altered by the patterning of individual- and couple-level risk and resilience factors for SS-IPV. These goals will be achieved by using the complementary strengths of laboratory-based experimental (Study 1) and longitudinal daily diary methods (Study 2), which are gold standard methods for establishing temporal relations among risk factors and IPV perpetration. For each study, we will recruit an independent sample of cisgender male-male and female-female couples who identify as gay or lesbian and are at high risk for IPV (based upon prior history of IPV) from Atlanta, GA. Across both studies, effects will be examined within an Actor-Partner Interdependence Modeling framework which will allow for valid analysis of both partners’ intersecting identities as well as risk and resilience factors at the individual- and couple-level. The most important contribution of the proposed project will be to provide the first comprehensive etiological model for SS-IPV perpetration that attends to both interactional and process-based factors that account for sexual minority stress and proximal alcohol effects at the individual- and couple-level of analysis. In doing so, results derived from this project will make a major contribution toward the evidence base required to develop effective, culturally-informed SS-IPV treatment and prevention efforts.