VASCULATA-2012

血管-2012

基本信息

  • 批准号:
    8397833
  • 负责人:
  • 金额:
    $ 0.5万
  • 依托单位:
  • 依托单位国家:
    美国
  • 项目类别:
  • 财政年份:
    2012
  • 资助国家:
    美国
  • 起止时间:
    2012-07-15 至 2013-06-30
  • 项目状态:
    已结题

项目摘要

DESCRIPTION (provided by applicant): Vasculata-2012 will be held in Nashville, Tennessee from July 25-28, 2012 with Vanderbilt University School of Medicine serving as the lead host institution in conjunction with University of Kentucky College of Medicine and Meharry Medical College. Vasculata-2012 will fulfill the following specific aims: Aim 1: Inspire young investigator from a variety of disciplines to enter the field of vascular biology by providing trainees with an introductory learning experience. Aim 2: Orient new investigators to the breadth and depth of the frontiers of knowledge of vascular biology through state-of-the-art presentations and scientific interchange with leading investigators. Aim 3: Enhancing exposure and understanding of the latest experimental approaches employed in vascular biology by a combination of didactic lectures and hands-on workshops. Aim 4: To allow young investigators to interact with established scientists in vascular biology via poster presentations and informal interactions. The Vasculata-2012 conference will be a distinctive addition to the Vasculata series by: 1) providing a wide range of relevant research topics ranging from basic concepts to clinical research, 2) introducing attendees to fields newly recognized to impact vascular biology, 3) providing workshops that will provide the trainees with firsthand experience with methods relevant to vascular biology. The faculty for Vasculata 2012 consists of core faculty from the three sponsoring institutions and carefully selected experts from other institutions. These faculty members include investigators from varied disciplines including developmental biology, matrix biology, vascular immunology, clinical science and genetic epidemiology. Vasculata 2012 will also emphasize new information regarding vascular diseases in minorities and women, and emerging vascular diseases in the third world. Overall, the Vasculata-2012 conference promises to be an exciting and uniquely rich research training experience. PUBLIC HEALTH RELEVANCE: The public health and clinical impact of vascular diseases such as hypertension, stroke and coronary heart disease are the major causes of death and disability in our nation. The scientific discipline of vascular biology remains a relatively young field with a small critica mass of scientific experts trained in the discipline. The Vasculata research training conference series serves as a critical national resource to the scientific community by providing an invaluable forum for research training and career development for the next generation of biomedical scientists in vascular biology and related fields.
描述(由申请人提供):Vasculata-2012将于2012年7月25日至28日在田纳西州纳什维尔举行,范德比尔特大学医学院将与肯塔基大学医学院和梅哈里医学院一起担任牵头主办机构。Vasculata-2012将实现以下具体目标:目标1:通过为学员提供入门学习体验,激励来自不同学科的年轻研究人员进入血管生物学领域。目标2:通过最先进的陈述和与领先研究人员的科学交流,将新的研究人员定向到血管生物学知识前沿的广度和深度。目标3:通过授课讲座和实践工作坊相结合的方式,增加对血管生物学最新实验方法的接触和理解。目标4:让年轻的研究人员通过海报展示和非正式互动与血管生物学领域的知名科学家互动。Vasculata-2012会议将是Vasculata系列的一个独特的补充:1)提供从基本概念到临床研究的广泛相关研究主题;2)向与会者介绍新认识到的影响血管生物学的领域;3)提供研讨会,为学员提供与血管生物学相关的方法的第一手经验。Vasculata 2012的教员由来自三个赞助机构的核心教员和来自其他机构的精心挑选的专家组成。这些教职员工 成员包括来自不同学科的研究人员,包括发育生物学、基质生物学、血管免疫学、临床科学和遗传流行病学。Vasculata 2012还将强调有关少数民族和妇女的血管疾病以及第三世界新出现的血管疾病的新信息。总体而言,Vasculata-2012大会有望成为一次令人兴奋和独特的丰富的研究培训体验。公共卫生相关性:高血压、中风和冠心病等血管疾病的公共健康和临床影响是我国死亡和残疾的主要原因。血管生物学的科学学科仍然是一个相对年轻的领域,只有一小批受过该学科培训的科学专家。Vasculata研究培训会议系列为科学界提供了一个宝贵的论坛,为血管生物学和相关领域的下一代生物医学科学家提供研究培训和职业发展的宝贵论坛,是科学界的重要资源。

项目成果

期刊论文数量(0)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)

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David G Harrison其他文献

Effects of Interleukin-1β Inhibition on Blood Pressure, Incident Hypertension, and Residual Inflammatory Risk
IL-1β 抑制对血压、高血压和残余炎症风险的影响
  • DOI:
  • 发表时间:
    2019
  • 期刊:
  • 影响因子:
    8.3
  • 作者:
    A. Rothman;Jean G Macfadyen;T. Thuren;A. Webb;David G Harrison;T. Guzik;P. Libby;R. Glynn;P. Ridker
  • 通讯作者:
    P. Ridker
Posttranscriptional Regulation of Endothelial Nitric Oxide Synthase during Cell Growth Materials and Methods Cell Culture
细胞生长过程中内皮一氧化氮合酶的转录后调节 材料和方法 细胞培养
  • DOI:
  • 发表时间:
  • 期刊:
  • 影响因子:
    0
  • 作者:
    Charles D. Searles;Yoichi Miwa;David G Harrison;S. Ramasamy;D. Searles
  • 通讯作者:
    D. Searles
Isolevuglandins promote autoimmunity and hypertension in systemic lupus erythematosus
异黄兰素促进系统性红斑狼疮的自身免疫和高血压
  • DOI:
    10.1101/2020.02.10.20021741
  • 发表时间:
    2020
  • 期刊:
  • 影响因子:
    0
  • 作者:
    David M. Patrick;N. Visitación;M. Ormseth;C. M. Stein;Sean S. Davies;Valery N. Yermalitsky;V. Amarnath;Leslie J. Crofford;Jonathan M Williams;S. Dikalov;A. Dikalova;Liang Xiao;Justin P. Van Beusecum;Mingfang Ao;A. Fogo;Kirabo Annet;David G Harrison
  • 通讯作者:
    David G Harrison
88 - The Redox Sensitive Inactivation of Mitochondrial Sirt3, SOD2 Hyperacetylation and Complex I Superoxide Production in Endothelial Dysfunction and Hypertension
  • DOI:
    10.1016/j.freeradbiomed.2015.10.128
  • 发表时间:
    2015-10-01
  • 期刊:
  • 影响因子:
  • 作者:
    Anna E Dikalova;Hanna A Itani;Rafal R Nazarewicz;William G McMaster;Joshua P Fessel;Jorge L Gamboa;David G Harrison;Sergey Dikalov
  • 通讯作者:
    Sergey Dikalov
A dynamical neural simulation mapping feature-based attention to location with non-linear cortical circuits
  • DOI:
    10.1186/1471-2202-12-s1-p55
  • 发表时间:
    2011-07-18
  • 期刊:
  • 影响因子:
    2.300
  • 作者:
    David G Harrison;Marc de Kamps
  • 通讯作者:
    Marc de Kamps

David G Harrison的其他文献

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{{ truncateString('David G Harrison', 18)}}的其他基金

Common Inflammation Pathways between Aging and Hypertension That Weaken Bone
衰老和高血压之间削弱骨骼的常见炎症途径
  • 批准号:
    10430633
  • 财政年份:
    2022
  • 资助金额:
    $ 0.5万
  • 项目类别:
Common Inflammation Pathways between Aging and Hypertension That Weaken Bone
衰老和高血压之间削弱骨骼的常见炎症途径
  • 批准号:
    10618349
  • 财政年份:
    2022
  • 资助金额:
    $ 0.5万
  • 项目类别:
Vanderbilt Hypertension and Blood Pressure Regulation Program
范德比尔特高血压和血压调节计划
  • 批准号:
    10385839
  • 财政年份:
    2019
  • 资助金额:
    $ 0.5万
  • 项目类别:
Vanderbilt Hypertension and Blood Pressure Regulation Program
范德比尔特高血压和血压调节计划
  • 批准号:
    10597621
  • 财政年份:
    2019
  • 资助金额:
    $ 0.5万
  • 项目类别:
Mechanisms of Immune Activation in Hypertension
高血压的免疫激活机制
  • 批准号:
    10543181
  • 财政年份:
    2018
  • 资助金额:
    $ 0.5万
  • 项目类别:
Mechanisms of Immune Activation in Hypertension
高血压的免疫激活机制
  • 批准号:
    10328922
  • 财政年份:
    2018
  • 资助金额:
    $ 0.5万
  • 项目类别:
Mechanisms of T cell Activation in Hypertension
高血压中 T 细胞激活的机制
  • 批准号:
    9978625
  • 财政年份:
    2016
  • 资助金额:
    $ 0.5万
  • 项目类别:
ADMINISTRATIVE & BIOSTATICAL CORE
行政的
  • 批准号:
    9978623
  • 财政年份:
    2016
  • 资助金额:
    $ 0.5万
  • 项目类别:
The Role of Inflammation in Cardiovascular Disease
炎症在心血管疾病中的作用
  • 批准号:
    9978598
  • 财政年份:
    2016
  • 资助金额:
    $ 0.5万
  • 项目类别:
The Role of The T Cell In The Genesis of Hypertension
T 细胞在高血压发生中的作用
  • 批准号:
    9273740
  • 财政年份:
    2015
  • 资助金额:
    $ 0.5万
  • 项目类别:

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