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Integrated bacterial genomics and virulence analysis of uropathogenic Streptococcus agalactiae

Integrated bacterial genomics and virulence analysis of uropathogenic Streptococcus agalactiae
尿路致病性无乳链球菌的综合细菌基因组学和毒力分析
批准号:
nhmrc : GNT1146820
负责人:
A/Pr Scott Beatson
金额:
$73.71万
依托单位:
依托单位国家:
澳大利亚
项目类别:
Project Grants
财政年份:
2018
资助国家:
澳大利亚
项目状态:
已结题
起止时间:
2018-01-01 至 --

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中文摘要
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英文摘要
Urinary tract infections (UTI), which start as a bladder infection and often evolve to encompass the kidneys, are among the most common infectious diseases in humans. Streptococcus agalactiae is an important cause of gram-positive bacterial UTI. We will study the genomes and functions of specific genes in reference strains of this bacterium isolated from patients with different forms of infection to elucidate how bacterial genes and virulence factors contribute to these types of infections.
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Integrated bacterial genomics and virulence analysis of uropathogenic Streptococcus agalactiae
  • 批准号:
    nhmrc : 1146820
  • 项目类别:
    Project Grants
  • 资助金额:
    $49.84万
  • 财政年份:
    2018
  • 负责人:
    A/Pr Scott Beatson
  • 依托单位:
Bacterial Pathogenomics: whole-genome sequencing to investigate infection transmission, pathogenesis and antibiotic resistance
  • 批准号:
    nhmrc : 1090456
  • 项目类别:
    Career Development Fellowships
  • 资助金额:
    $31.74万
  • 财政年份:
    2015
  • 负责人:
    A/Pr Scott Beatson
  • 依托单位:
Bacterial Pathogenomics: whole-genome sequencing to investigate infection transmission, pathogenesis and antibiotic resistance
  • 批准号:
    nhmrc : GNT1090456
  • 项目类别:
    Career Development Fellowships
  • 资助金额:
    $45.55万
  • 财政年份:
    2015
  • 负责人:
    A/Pr Scott Beatson
  • 依托单位:
Functional and genomic analysis of E. coli that cause urinary tract infection
  • 批准号:
    nhmrc : GNT1067455
  • 项目类别:
    Project Grants
  • 资助金额:
    $79.31万
  • 财政年份:
    2014
  • 负责人:
    A/Pr Scott Beatson
  • 依托单位:
国内基金
海外基金
中国棉铃虫核多角体病毒基因组库和分子进化
  • 批准号:
    30540076
  • 项目类别:
    专项基金项目
  • 资助金额:
    8.0万元
  • 批准年份:
    2005
  • 负责人:
    王汉中
  • 依托单位:
细菌脂蛋白(BLP)诱导LPS交叉耐受的分子机理研究