2017 Glial Biology Gordon Research Conference & Gordon Research Seminar
2017 Glial Biology Gordon Research Conference & Gordon Research Seminar
批准号:
9331201
负责人:
Beth Ann Stevens
金额:
$1.0万
依托单位国家:
美国
项目类别:
财政年份:
2017
资助国家:
美国
项目状态:
已结题
起止时间:
2017-03-01 至 2018-02-28
关键词:
AmericasAsiaAutistic DisorderBiological ModelsBiologyBrainBrain DiseasesCell physiologyCellular biologyCollaborationsCommunitiesDevelopmentDisciplineDiseaseEuropeExposure toFosteringFunctional disorderFundingGoalsGrantHeterogeneityHuntington DiseaseInstitutesInternationalMentorshipMetabolicMissionNational Institute of Mental HealthNational Institute of Neurological Disorders and StrokeNervous System PhysiologyNervous system structureNeurodegenerative DisordersNeurodevelopmental DisorderNeurogliaNeurosciencesPostdoctoral FellowResearchResearch PersonnelRoleScientistShapesSyndromeSystems BiologyTechnologyUnderrepresented MinorityUnited States National Institutes of HealthVocational GuidanceWorkcell typedisabilityglial cell developmentgraduate studentin vivomeetingsnervous system developmentneural circuitneuropsychiatric disordernew technologypostersprogramssuccesssymposiumtraining opportunityunpublished works
中文摘要
摘要
神经胶质生物学的GRC将于2017年举办第8次会议。吸引了来自欧洲,亚洲,
和美洲,它被认为是最重要的胶质细胞国际会议之一,
神经胶质生物学作为神经科学的一个新兴分支的出现。GRC对神经胶质细胞的总体目标
生物学是分享和讨论尖端研究集中在神经胶质细胞的发展,异质性和
神经系统中胶质细胞和其他细胞类型之间的功能相互作用。2017年会议将在
强调神经胶质在大脑发育中的作用,包括神经胶质在神经回路形成和可塑性中的作用,
以及神经胶质细胞功能障碍对神经发育和神经退行性疾病的作用
包括孤独症、瑞特综合症和亨廷顿氏病。本次会议还将突出国家的最先进的
这些技术将有助于更好地了解体内神经胶质细胞的发育和功能。42
发言人和9名代表成熟和年轻研究人员的讨论领导人承诺,
参加这笔赠款的支持对于最大限度地提高受训者的参与至关重要,因为它部分资助了戈登
神经胶质生物学研究研讨会,将在GRC之前举办第二次会议。GRS将提供
为该领域的年轻科学家(研究生和博士后)提供一个聚会、分享想法、接受
导师从选定的既定研究者,并介绍他们的工作进展,无论是通过平台或
海报展示。重要的是,包括来自所有人群的调查人员,包括代表性不足的人,
少数民族和残疾人将是最优先事项。
在这两个GRC和GRS,指定的会议将集中在神经胶质细胞的发展和功能,
健康和患病的大脑以及更广泛的神经系统中的回路和代谢支持。的
传播尖端研究成果,为初级教育提供广泛的培训机会
这次会议使科学家们对国家卫生研究院的总体使命高度相关。
英文摘要
ABSTRACT
The GRC on glial biology will host its 8th meeting in 2017. Drawing together investigators from Europe, Asia
and the Americas, it is considered one of the foremost international meetings on glia and is credited with the
emergence of glial biology as a burgeoning subfield of Neuroscience. The overarching aim of the GRC on glial
biology is to share and discuss cutting edge research focused on glial cell development, heterogeneity and
functional interactions between glia and other cell types in the nervous system. The 2017 meeting will place
emphasis on the role of glia in brain development, including glial roles in neural circuit formation and plasticity,
and also the contribution of glial cell dysfunction to neurodevelopmental and neurodegenerative disorders
including autism, Rhett syndrome, and Huntington’s disease. This meeting will also highlight state-of-the-art
technologies that will facilitate a better understanding of glial cell development and function in vivo. 42
speakers and 9 discussion leaders representing both established and young investigators have committed to
participate. The support of this grant is critical to maximize trainee participation, as it in part funds the Gordon
Research Seminar on Glial Biology, which will host its 2nd meeting just prior to the GRC. The GRS will provide
an opportunity for young scientists (graduate students and postdocs) in the field to meet, share ideas, receive
mentorship from select established investigators, and present their works in progress either via platform or
poster presentations. Importantly, inclusion of investigators from all people groups, including underrepresented
minorities and those with disabilities, will be a top priority.
In both the GRC and GRS, designated sessions will focus on the development and function of glia in neural
circuitry and metabolic support in both the healthy and diseased brain and the broader nervous system. The
dissemination of cutting edge research findings and extensive training opportunities for junior
scientists enabled by this meeting are highly pertinent to the overall mission of the NIH.
期刊论文(0)
专著(0)
科研奖励(0)
会议论文
Mechanisms of Activity-dependent Microglia-neuron Interactions in Development and Disease
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Investigating CD47-SIRPa as novel protective signals during CNS synaptic pruning
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批准号:8937448
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项目类别:
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Mechanisms of activity-dependent microglia-neuron interactions in development and disease
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Mechanisms of activity-dependent microglia-neuron interactions in development and disease
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批准号:10612683
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项目类别:
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负责人:Beth Ann Stevens
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依托单位:
Complement-Microglia Interaction in Synaptic Loss and Neurodegeneration in HD
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批准号:9151051
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项目类别:
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负责人:Beth Ann Stevens
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依托单位:
Complement-Microglia Interaction in Synaptic Loss and Neurodegeneration in HD
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负责人:Beth Ann Stevens
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Complement-Microglia Interaction in Synaptic Loss and Neurodegeneration in HD
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项目类别:
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负责人:Beth Ann Stevens
-
依托单位:
Complement-Microglia Interaction in Synaptic Loss and Neurodegeneration in HD
-
批准号:9099977
-
项目类别:
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依托单位:
Investigating the Role of Glia in Activity-Dependent Synapse Elimination
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批准号:8644327
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项目类别:
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资助金额:$37.68万
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Investigating the Role of Glia in Activity-Dependent Synapse Elimination
-
批准号:8468223
-
项目类别:
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资助金额:$36.73万
-
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负责人:Beth Ann Stevens
-
依托单位:
Investigating the Role of Glia in Activity-Dependent Synapse Elimination
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批准号:8238593
-
项目类别:
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资助金额:$36.41万
-
财政年份:2011
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负责人:Beth Ann Stevens
-
依托单位:
Investigating the Role of Glia in Activity-Dependent Synapse Elimination
-
批准号:8846681
-
项目类别:
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资助金额:$38.06万
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财政年份:2011
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负责人:Beth Ann Stevens
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依托单位:
Investigating the Role of Glia in Activity-Dependent Synapse Elimination
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批准号:9061442
-
项目类别:
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资助金额:$38.06万
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财政年份:2011
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负责人:Beth Ann Stevens
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依托单位:
Investigating the Role of Glia in Activity-Dependent Synapse Elimination
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批准号:8320868
-
项目类别:
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资助金额:$38.06万
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依托单位:
How is the Blood-Brain Barrier Formed?
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项目类别:
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依托单位:
How is the Blood-Brain Barrier Formed?
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批准号:6847421
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项目类别:
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资助金额:$4.4万
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依托单位:
Administrative Core - Core A
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批准号:9923736
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项目类别:
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资助金额:$18.81万
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财政年份:--
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依托单位:
Administrative Core - Core A
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批准号:9280282
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项目类别:
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财政年份:--
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负责人:Beth Ann Stevens
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依托单位:
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