Phagosomal Signals Shape Inflammatory Responses to Borrelia Burgdorferi

吞噬体信号塑造对伯氏疏螺旋体的炎症反应

• 批准号: 8145100
• 负责人: Juan C Salazar
• 金额: $ 41.56万
• 依托单位: UNIVERSITY OF CONNECTICUT SCH OF MED/DNT
• 依托单位国家: 美国
• 财政年份: 2010
• 资助国家: 美国
• 起止时间: 2010-09-27 至 2011-08-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/8145100
• 关键词: Address; Anti-Inflammatory Agents; Anti-inflammatory; Area; Bacteria; Borrelia burgdorferi; CD14 gene; Castor Bean Tick; Cell surface; Cells; Clinical; Communicable Diseases; Complex; Deer; Disease; Elements; Exotoxins; Genes; Genetic Transcription; Goals; Growth; Hand; Human; Immune; Immune response; In Vitro; Inflammatory; Inflammatory Response; Interferon Type I; Interferons; Invaded; Knockout Mice; Laboratories; Life; Lipoproteins; Lyme Disease; Mediating; Membrane; Modeling; Mus; Order Spirochaetales; Orthologous Gene; Patients; Phagocytosis; Phagosomes; Population; Process; Reporting; Research; Research Personnel; Shapes; Signal Transduction; Signs and Symptoms; Surface; System; TLR8 gene; Tail; Ticks; Tissues; Toll-Like Receptor 1; Toll-like receptors; United States; Vacuole; base; cytokine; cytosolic receptor; extracellular; in vivo; insight; macrophage; monocyte; mouse model; pathogen; public health relevance; research study; response; vector