Effects of intestinal microbes on rheumatic diseases: A bibliometric analysis.
Effects of intestinal microbes on rheumatic diseases: A bibliometric analysis.
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DOI：
10.3389/fmicb.2022.1074003
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发表时间：
2022
影响因子：
5.2
通讯作者：
Ji S
中科院分区：
文献类型：
--
作者：
Huang R;Zhang M;Lu Y;Xu D;Liu Y;Jin M;Xian S;Wang S;Tong X;Lu J;Zhang W;Qian W;Tang J;Yang Y;Lu B;Chang Z;Liu X;Ji S
风湿性疾病是一组多系统炎性自身免疫性疾病，其病因尚不清楚。在过去的几十年里，研究人员发现了风湿性疾病与肠道微生物群之间存在关联的痕迹，这可以部分解释风湿性疾病的发病机制。本文综述了肠道植物群与风湿性疾病相互作用的研究动态和主要分类，并探讨了其可能的临床应用。我们分析了Web of Science核心收藏（日期为2022年5月15日）的文献计量数据。使用Biblioshiny R语言软件包（bibliomektop）获得年度出版物和引文，根据布拉德福德定律获得核心来源，以及国家合作地图。借助VOSviewer和CiteSpace设计并验证了关键词共现网络和策略图，将研究主题细分为若干主题，确定了研究维度。大多数本地引用文献和核心来源的表格都是手工处理的。此外，Altmetric注意力得分和年度Altmetric前100名被用来分析年度出版物和引文。从共541篇文献中发现，年度发表和被引的总体趋势是增加的。主要研究方法是比较某些风湿性疾病患者和对照组的肠道微生物组成，以确定与疾病发生和发展相关的微生物变化。根据布拉德福德定律，核心来源是关节炎和风湿病学，风湿病年鉴，风湿病学最新观点，营养素，风湿病学和风湿病学杂志。自1976年以来，已有101个国家或地区参与了血液流变学和肠道微生物的研究。美国位居榜首，与其他国家有着最广泛的学术联系。主要包括肠道细菌引起的炎症在风湿病发病机制中的关键作用、风湿病与炎症性肠病的密切关系、风湿病与肠道植物群相互作用的免疫调节机制、风湿病患者肠道菌群失调和多样性降低以及口腔植物群对风湿病的影响等5个方面。此外，还确定了四个研究维度，包括病理学、治疗、疾病和实验。对风湿性疾病和肠道微生物群的研究正在增长。应注意它们之间相互作用的机制，如微生物-免疫-RD串扰。希望本研究成果能应用于疾病的预防、诊断和治疗，并为读者今后的研究工作做出贡献。
Rheumatic diseases (RD) are a group of multi-system inflammatory autoimmune diseases whose causes are still under study. In the past few decades, researchers have found traces of the association between rheumatic diseases and intestinal microbiota, which can partially explain the pathogenesis of rheumatic diseases. We aimed to describe the research trend and main divisions on how gut flora interreacts with rheumatic diseases, and discussed about the possible clinical applications. We analyzed bibliometric data from the Web of Science core collection (dated 15th May 2022). Biblioshiny R language software packages (bibliometrix) were used to obtain the annual publication and citations, core sources according to Bradford’s law, and country collaboration map. We designed and verified the keyword co-occurrence network and strategic diagram with the help of VOSviewer and CiteSpace, subdivided the research topic into several themes and identified research dimensions. The tables of most local cited documents and core sources were processed manually. Furthermore, the Altmetric Attention Score and the annual Altmetric Top 100 were applied to analyze the annual publication and citation. From a total of 541 documents, we found that the overall trend of annual publication and citation is increasing. The major research method is to compare the intestinal microbial composition of patients with certain rheumatic disease and that of the control group to determine microbial alterations related to the disease’s occurrence and development. According to Bradford’s law, the core sources are Arthritis and Rheumatology, Annals of the Rheumatic Diseases, Current Opinion in Rheumatology, Nutrients, Rheumatology, and Journal of Rheumatology. Since 1976, 101 countries or regions have participated in studies of rheumatology and intestinal microbes. The United States ranks at the top and has the broadest academic association with other countries. Five themes were identified, including the pivotal role of inflammation caused by intestinal bacteria in the rheumatic pathogenesis, the close relationship between rheumatic diseases and inflammatory bowel disease, immunoregulation mechanism as a mediator of the interaction between rheumatic diseases and gut flora, dysbiosis and decreased diversity in intestine of patients with rheumatic diseases, and the influence of oral flora on rheumatic diseases. Additionally, four research dimensions were identified, including pathology, treatment, disease, and experiments. Studies on rheumatic diseases and the intestinal microbiota are growing. Attention should be paid to the mechanism of their interaction, such as the microbe-immune-RD crosstalk. Hopefully, the research achievements can be applied to diseases’ prevention, diagnosis, and treatment, and our work can contribute to the readers’ future research.
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影响因子：
--
作者：
Crowson, Cynthia S.;Matteson, Eric L.;Myasoedova, Elena;Michet, Clement J.;Ernste, Floranne C.;Warrington, Kenneth J.;Davis, John M., III;Hunder, Gene G.;Therneau, Terry M.;Gabriel, Sherine E.
通讯作者：
Gabriel, Sherine E.
影响因子：
4.6
作者：
Guo Z;Zhang J;Wang Z;Ang KY;Huang S;Hou Q;Su X;Qiao J;Zheng Y;Wang L;Koh E;Danliang H;Xu J;Lee YK;Zhang H
通讯作者：
Zhang H
影响因子：
64.8
作者：
Chassaing B;Koren O;Goodrich JK;Poole AC;Srinivasan S;Ley RE;Gewirtz AT
通讯作者：
Gewirtz AT
影响因子：
27.4
作者：
Breban, Maxime;Tap, Julien;Sokol, Harry
通讯作者：
Sokol, Harry
影响因子：
27.4
作者：
Cross, Marita;Smith, Emma;March, Lyn
通讯作者：
March, Lyn