Dissertation Research: EXPLORING THE DIFFERENTIAL UPTAKE AND BREAKDOWN OF PHOSPHORUS BY FRESHWATER BACTERIAL POPULATIONS

人类活动加速了地表淡水的富营养化，这是一个有据可查但复杂且知之甚少的现象。淡水富营养化导致全球生态系统服务和水质严重恶化，自19世纪末S以来一直是威斯康星州中部湖泊的一个持续问题。限制性营养物质的增加，特别是磷，被认为是加速富营养化的主要触发因素。微生物(主要是藻类和细菌)被认为控制着湖泊中的磷循环，但对推动这一过程的微生物参与者的详细了解尚不清楚。在这项提议中，研究人员的目标是确定参与威斯康星州富营养化湖泊中磷循环的主要细菌种群。识别参与含磷化合物吸收和分解的细菌将有助于识别推动湖泊磷循环的机制，并将提高根据政策变化预测水质的能力。

The accelerated eutrophication of surface freshwater due to human activity is a well-documented, but complex and poorly understood phenomenon. Freshwater eutrophication causes serious deterioration of ecosystem services and water quality worldwide and has been a persistent problem in central Wisconsin lakes since the late 1800's. Increased amounts of limiting nutrients, especially phosphorus, are considered the primary trigger for accelerated eutrophication. Microbes (primarily algae and bacteria) are thought to control the phosphorus cycle in lakes, yet a detailed understanding of the microbial participants driving this process is unknown. In this proposal the researchers aim to identify the primary bacterial populations that are involved in phosphorus cycling within eutrophic Wisconsin lakes. Identification of bacteria involved in both the uptake and breakdown of phosphorus containing compounds will aid in the identification of the mechanisms driving the phosphorus cycle in lakes and will lead to an improved ability to predict water quality in response to policy changes.