Increased pest resistance in oilseed rape mediated by an enhanced UV-B response

通过增强的 UV-B 响应增强油菜的害虫抗性

• 批准号: BB/I017518/1
• 负责人: Gareth Islwyn Jenkins
• 金额: $ 48.12万
• 依托单位: University of Glasgow
• 依托单位国家: 英国
• 项目类别: Research Grant
• 财政年份: 2011
• 资助国家: 英国
• 起止时间: 2011 至 无数据
• 项目状态: 已结题
• 来源: https://gtr.ukri.org/projects?ref=BB%2FI017518%2F1
• 关键词: Increased; pest; resistance; oilseed; rape

Attack by insects, molluscs and microbial pathogens causes significant losses to the oilseed rape crop and considerable expense is incurred in trying to counter this problem by using pesticides. The aim of this project is to undertake research to underpin the development of new oilseed rape varieties with increased resistance to insect herbivory. Exposure of plants to UV-B wavelengths, which are a natural component of sunlight, has been shown to reduce the attractiveness of plants to insect herbivores by altering plant chemical composition. Several plant responses to UV-B are mediated by a protein called UVR8 and the defence response to herbivory involves the regulatory molecule jasmonic acid. In this project, we will examine the potential of UVR8 and a protein called MYC2 that mediates the response to jasmonic acid to increase resistance to insect herbivory in oilseed rape. We will test whether transgenic over-expression of specific genes increases resistance to insect herbivory as 'proof of concept' that manipulation of the UV-B response could be used to develop new varieties.

昆虫，软体动物和微生物病原体的攻击会对石油种子作物造成重大损失，并试图通过使用农药来解决这一问题。该项目的目的是进行研究，以增强对昆虫食草动物的耐药性的发展发展。植物接触到阳光的自然成分的UV-B波长，已被证明可以通过改变植物化学成分来降低植物对食草动物的吸引力。几种植物对UV-B的反应是由一种称为UVR8的蛋白质介导的，对草食性的防御反应涉及调节分子茉莉酸。在这个项目中，我们将检查UVR8的潜力和一种称为MYC2的蛋白质，该蛋白质介导对茉莉酸的反应，以增加油料种子强奸中对昆虫食草动物的抗性。我们将测试特定基因的转基因过表达是否会增加对昆虫草食性的抗性，因为“概念证明”可以使用对UV-B反应的操纵来开发新品种。