IMPLICATIONS OF DISULFIDES BREAKING SEQUENCE IN REDUCTIVE UNFOLDING PATHWAYS

二硫化物断裂序列对还原性展开路径的影响

基本信息

  • 批准号:
    7721227
  • 负责人:
  • 金额:
    $ 0.2万
  • 依托单位:
  • 依托单位国家:
    美国
  • 项目类别:
  • 财政年份:
    2008
  • 资助国家:
    美国
  • 起止时间:
    2008-05-15 至 2009-03-31
  • 项目状态:
    已结题

项目摘要

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The sequence of the breakage of disulfide bonds due to x-ray irradiation was observed by time-dependent crystallographic studies of proteins using the strategy we developed for time-dependent structure observations on the atomic scale in a protein crystal for standard crystallography experiments. These observed sequences are related to the reductive unfolding pathways in proteins. he current study focuses on bovine pancreatic ribonuclease A, where the observed breaking sequence is being compared to results by other established methods.
这个子项目是众多研究子项目之一

项目成果

期刊论文数量(0)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)

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JUN WANG其他文献

JUN WANG的其他文献

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{{ truncateString('JUN WANG', 18)}}的其他基金

Elucidating the Role of Cancer-Associated FGL1 in Tumor Immunity and Developing FGL1-Guided Anti-LAG-3 Cancer Immunotherapy
阐明癌症相关 FGL1 在肿瘤免疫中的作用并开发 FGL1 引导的抗 LAG-3 癌症免疫疗法
  • 批准号:
    10504399
  • 财政年份:
    2022
  • 资助金额:
    $ 0.2万
  • 项目类别:
Elucidating the Immune Suppressive Mechanism of SIGLEC-15 in the Tumor Microenvironment
阐明 SIGLEC-15 在肿瘤微环境中的免疫抑制机制
  • 批准号:
    10587743
  • 财政年份:
    2022
  • 资助金额:
    $ 0.2万
  • 项目类别:
Elucidating the Role of Cancer-Associated FGL1 in Tumor Immunity and Developing FGL1-Guided Anti-LAG-3 Cancer Immunotherapy
阐明癌症相关 FGL1 在肿瘤免疫中的作用并开发 FGL1 引导的抗 LAG-3 癌症免疫疗法
  • 批准号:
    10663382
  • 财政年份:
    2022
  • 资助金额:
    $ 0.2万
  • 项目类别:
Functional characterization of SARS-CoV-2 myeloid cell receptors as an immunopathogenic mechanisms of COVID-19
SARS-CoV-2 骨髓细胞受体作为 COVID-19 免疫致病机制的功能特征
  • 批准号:
    10288857
  • 财政年份:
    2021
  • 资助金额:
    $ 0.2万
  • 项目类别:
Functional characterization of SARS-CoV-2 myeloid cell receptors as an immunopathogenic mechanisms of COVID-19
SARS-CoV-2 骨髓细胞受体作为 COVID-19 免疫致病机制的功能特征
  • 批准号:
    10443833
  • 财政年份:
    2021
  • 资助金额:
    $ 0.2万
  • 项目类别:
HIGH RESOLUTION STRUCTURES BY MINIMIZING RADIATION EFFECTS
通过最小化辐射效应实现高分辨率结构
  • 批准号:
    7721228
  • 财政年份:
    2008
  • 资助金额:
    $ 0.2万
  • 项目类别:
SPECIFIC CHEMICAL INTERACTIONS IN MATRIX-INCORPORATED INSULIN STRUCTURES
基质结合的胰岛素结构中的特定化学相互作用
  • 批准号:
    7721229
  • 财政年份:
    2008
  • 资助金额:
    $ 0.2万
  • 项目类别:
SPECIFIC CHEMICAL INTERACTIONS IN MATRIX-INCORPORATED INSULIN STRUCTURES
基质结合的胰岛素结构中的特定化学相互作用
  • 批准号:
    7369520
  • 财政年份:
    2005
  • 资助金额:
    $ 0.2万
  • 项目类别:
IMPLICATIONS OF DISULFIDES BREAKING SEQUENCE IN REDUCTIVE UNFOLDING PATHWAYS
二硫化物断裂序列对还原性展开路径的影响
  • 批准号:
    7369518
  • 财政年份:
    2005
  • 资助金额:
    $ 0.2万
  • 项目类别:
HIGH RESOLUTION STRUCTURES BY MINIMIZING RADIATION EFFECTS
通过最小化辐射效应实现高分辨率结构
  • 批准号:
    7369519
  • 财政年份:
    2005
  • 资助金额:
    $ 0.2万
  • 项目类别:

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