HIV-1 AND ENDOTHELIAL PROGENITOR CELLS

HIV-1 和内皮祖细胞

• 批准号: 7719562
• 负责人: Christopher J de Souza
• 金额: $ 0.22万
• 依托单位: UNIVERSITY OF COLORADO DENVER
• 依托单位国家: 美国
• 财政年份: 2008
• 资助国家: 美国
• 起止时间: 2008-04-01 至 2008-05-31
• 项目状态: 已结题
• 来源: https://reporter.nih.gov/project-details/7719562
• 关键词: Blood Vessels; Bone Marrow; Cell physiology; Cells; Computer Retrieval of Information on Scientific Projects Database; Endothelial Cells; Funding; Grant; HIV-1; Heart; Individual; Infection; Institution; Link; Numbers; Purpose; Research; Research Personnel; Resources; Risk; Source; Stem cells; United States National Institutes of Health; repaired

This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. Individuals with HIV-1 infection demonstrate an increased risk for heart attach. Endothelial cell progenitor cells are cells produced by bone marrow to help repair blood vessels. A reduction in the number and function of these cells have been linked to an increased risk of heart attach. The purpose of this study is to determine whether endothelial progenitor cells are reduced in individuals with HIV-1 infection.

这个子项目是许多研究子项目中的一个 由NIH/NCRR资助的中心赠款提供的资源。子项目和 研究者（PI）可能从另一个NIH来源获得了主要资金， 因此可以在其他CRISP条目中表示。所列机构为 研究中心，而研究中心不一定是研究者所在的机构。 HIV-1感染者显示心脏附连的风险增加。 内皮细胞祖细胞是骨髓产生的细胞，有助于修复血管。这些细胞的数量和功能的减少与心脏附着的风险增加有关。 本研究的目的是确定是否内皮祖细胞减少与HIV-1感染的个人。