Neuropsychological basic deficits and developmental pathways of attention-deficit/hyperactivity disorder: preschool to school age
注意力缺陷/多动障碍的神经心理学基本缺陷和发展途径:学龄前至学龄
基本信息
- 批准号:224833626
- 负责人:
- 金额:--
- 依托单位:
- 依托单位国家:德国
- 项目类别:Research Grants
- 财政年份:2012
- 资助国家:德国
- 起止时间:2011-12-31 至 2018-12-31
- 项目状态:已结题
- 来源:
- 关键词:
项目摘要
There is a broad consensus that executive and reward-related basic deficits emerging already in the preschool period characterize specific subtypes (developmental pathways) of attention deficit/hyperactivity disorder (ADHD). Moreover, emotional dysregulation seems to be an essential feature of these subtypes. It is further assumed that the executive and reward-related basic deficits moderate the effects of family adversity factors on the development of ADHD-symptoms between preschool and school-age. Empirical research on these issues, however, is scarce.Our research project has two objectives: (1) To analyze validity aspects of the assessment of the basic deficits at preschool-and school-age, including differentiation from emotional dysregulation. (2) To predict developmental trajectories of ADHD symptoms between preschool and school-age from basic deficits, environmental risks and the effects of their mutual interaction.A longitudinal study is conducted involving children with an increased risk for ADHD development and control children with assessment waves at the ages of four and five years. The assessment wave at school-age (eight years) is being planned. Results of the longitudinal study could add to current knowledge regarding early identification of children at risk for ADHD development and prevention of possible (secondary) maladjustment at school age.
一个广泛的共识是,在学龄前阶段已经出现的与执行和奖励相关的基本缺陷是注意力缺陷/多动障碍(ADHD)的特定亚型(发展途径)的特征。此外,情绪失调似乎是这些亚型的一个基本特征。我们进一步假设,在学龄前和学龄期之间,与执行和奖励相关的基本缺陷会缓和家庭逆境因素对ADHD症状发展的影响。然而,关于这些问题的实证研究还很少。我们的研究项目有两个目的:(1)分析学龄前和学龄期基本缺陷评估的效度,包括与情绪调节障碍的区别。(2)从基本缺陷、环境风险及其相互作用的影响预测ADHD症状在学龄前和学龄期之间的发展轨迹。学龄儿童(八岁)的评估浪潮正在计划中。这项纵向研究的结果可能会增加关于及早识别有ADHD发展风险的儿童和预防学龄儿童可能的(继发性)适应不良的现有知识。
项目成果
期刊论文数量(12)
专著数量(0)
科研奖励数量(0)
会议论文数量(0)
专利数量(0)
Psychosocial risk factors underlie the link between attention deficit hyperactivity symptoms and overweight at school entry
心理社会风险因素是注意力缺陷多动症状与入学时超重之间联系的基础
- DOI:10.1007/s00787-016-0870-1
- 发表时间:2017
- 期刊:
- 影响因子:6.4
- 作者:Pauli-Pott;Reinhardt;Wollenberg;Schroer;Heinzel-Gutenbrunner;Becker
- 通讯作者:Becker
Do different ADHD-related etiological risks involve specific neuropsychological pathways? An analysis of mediation processes by inhibitory control and delay aversion.
不同的 ADHD 相关病因风险是否涉及特定的神经心理学途径?
- DOI:10.1111/jcpp.12059
- 发表时间:2013
- 期刊:
- 影响因子:0
- 作者:Pauli-Pott;Mingebach;Roller;Becker
- 通讯作者:Becker
[Expressed emotion, mother-child relationship, and ADHD symptoms in preschool- a study on the validity of the German Preschool Five Minute Speech Sample].
【学前情绪表达、母子关系与多动症症状——德国学前五分钟言语样本效度研究】
- DOI:10.1024/1422-4917/a000384
- 发表时间:2015
- 期刊:
- 影响因子:0
- 作者:Schloß;Schramm;Christiansen;Scholz;Döpfner;Becker;Pauli-Pott
- 通讯作者:Pauli-Pott
Low hair cortisol concentration and emerging attention‐deficit/hyperactivity symptoms in preschool age
学龄前儿童头发皮质醇浓度低和出现注意力缺陷/多动症状
- DOI:10.1002/dev.21627
- 发表时间:2018
- 期刊:
- 影响因子:2.2
- 作者:Schloß;Müller;Becker;Skoluda;U. M. ;Pauli-Pott
- 通讯作者:Pauli-Pott
Attention deficit/hyperactivity and comorbid symptoms in preschoolers: Differences between subgroups in neuropsychological basic deficits
学龄前儿童的注意力缺陷/多动和共病症状:神经心理学基本缺陷亚组之间的差异
- DOI:10.1080/09297049.2013.778236
- 发表时间:2014
- 期刊:
- 影响因子:2.2
- 作者:Pauli-Pott;Mingebach;Roller;Becker
- 通讯作者:Becker
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Professorin Dr. Katja Becker其他文献
Professorin Dr. Katja Becker的其他文献
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{{ truncateString('Professorin Dr. Katja Becker', 18)}}的其他基金
Glucose 6-phosphate dehydrogenase 6-phosphogluconolactonase of the malaria parasite Plasmodium falciparum
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35551341 - 财政年份:2006
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Research Grants
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- 批准号:
5383507 - 财政年份:2002
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Research Grants
Human thioredoxin Reductases: Selenoproteins as Potential Drug Targets
人硫氧还蛋白还原酶:硒蛋白作为潜在的药物靶点
- 批准号:
5241800 - 财政年份:2000
- 资助金额:
-- - 项目类别:
Priority Programmes
Disulfidreduktasen und Glutathion-gestützter Metabolismus des Malaria-Erregers Plasmodium falciparum
疟疾病原体恶性疟原虫的二硫键还原酶和谷胱甘肽支持的代谢
- 批准号:
5128742 - 财政年份:1998
- 资助金额:
-- - 项目类别:
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