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Identification of functionally important autophosphorylation site(s) on ataxia telangiectasia and Rad 3 - related (ATR) protein kinase.

Identification of functionally important autophosphorylation site(s) on ataxia telangiectasia and Rad 3 - related (ATR) protein kinase.
共济失调毛细血管扩张症和 Rad 3 相关 (ATR) 蛋白激酶上功能重要的自磷酸化位点的鉴定。
批准号：
DP0666572
负责人：
Prof Martin Lavin
金额：
$18.34万
依托单位：
The University of Queensland
依托单位国家：
澳大利亚
项目类别：
Discovery Projects
财政年份：
2006
资助国家：
澳大利亚
项目状态：
已结题
起止时间：
2006-01-01 至 2009-09-30
来源：
https://dataportal.arc.gov.au/RGS/Web/Grants/DP0666572
关键词：
Identificationfunctionallyimportantautophosphorylationsite

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The integrity of our genetic material must be maintained so that it can be passed on from one generation to the next and also to minimize the risk of cancer and other pathologies in an individual. There are multiple proteins involved in protecting our DNA including several enzymes that detect and signal DNA damage to a series of pathways involved in halting the passage of cells through the cell cycle so that repair can occur. This project studies the mechanism of action of one of these enzymes which will be of benefit in designing new compounds to fight disease.
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