Pathogenic changes of pathogenic bacteriaby catecholamine

包括去甲肾上腺素（NE）在内的儿茶酚胺是细菌感染的促发因子。本研究的目的是阐明引起儿茶酚胺的致病菌的致病激活机制，以帮助感染性疾病的治疗。在本研究中，我们分析了使用副溶血性弧菌进行研究的申请人是一致的。副溶血性弧菌感染通常可被认为是肠道细菌感染的示例性模型。我们搜索的存在下，NE的受体在副溶血性弧菌的表面上。并进一步分析了NE在副溶血性弧菌中的调控机制和致病基因的表达。.

Catecholamine, including norepinephrine (NE), has been known as precipitating factor in bacterial infection. The aim of this study was to clarify the pathogenic activation mechanism of pathogenic bacteria that cause the catecholamine, to help infectious diseases treatment.In this study, we analyzed using Vibrio parahaemolyticushave been studying the applicant is consistently. V.parahaemolyticusinfection can be thought of as an exemplary model of intestinal bacterial infections in general. We searched for the presence of receptors for NE on the surface in V.parahaemolyticus. And we analyzedof the regulatory mechanism and expression pathogenic gene by the NE furtherin V.parahaemolyticus. .