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EARLY IMMUNE EVENTS IN CHILDHOOD ASTHMA

EARLY IMMUNE EVENTS IN CHILDHOOD ASTHMA
儿童哮喘的早期免疫事件
批准号：
8172554
负责人：
LISA M MILLER
金额：
$11.41万
依托单位：
UNIVERSITY OF CALIFORNIA AT DAVIS
依托单位国家：
美国
项目类别：
财政年份：
2010
资助国家：
美国
项目状态：
已结题
起止时间：
2010-06-01 至 2011-04-30
来源：
https://reporter.nih.gov/project-details/8172554
关键词：
Childhood AsthmaClinicalComputer Retrieval of Information on Scientific Projects DatabaseDevelopmentEventFundingGrantImmuneInstitutionLungMacaca mulattaModelingPulmonary EosinophiliaResearchResearch PersonnelResourcesSourceSymptomsUnited States National Institutes of Healthallergic airway diseasepostnatal

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中文摘要
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英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The primary objective of this project is to determine the early pulmonary mechanisms that initiate development of clinical symptoms in childhood asthma, using a rhesus monkey model of allergic airways disease. We have hypothesized that pulmonary eosinophilia is a critical first step in the initiation of allergic airways disease during postnatal development.
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The Role of Copper in Cerebral Amyloid Angiopathy
  • 批准号：
    9919005
  • 项目类别：
  • 资助金额：
    $38.38万
  • 财政年份：
    2017
  • 负责人：
    LISA M MILLER
  • 依托单位：
    UNIVERSITY OF RHODE ISLAND
The Role of Copper in Cerebral Amyloid Angiopathy
The Role of Copper in Cerebral Amyloid Angiopathy
ROLE OF EPITHELIUM IN AIRWAY IMMUNITY
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