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EARLY IMMUNE EVENTS IN CHILDHOOD ASTHMA

EARLY IMMUNE EVENTS IN CHILDHOOD ASTHMA
儿童哮喘的早期免疫事件
批准号：
7959049
负责人：
LISA M MILLER
金额：
$10.67万
依托单位：
UNIVERSITY OF CALIFORNIA AT DAVIS
依托单位国家：
美国
项目类别：
财政年份：
2009
资助国家：
美国
项目状态：
已结题
起止时间：
2009-05-01 至 2010-04-30
来源：
https://reporter.nih.gov/project-details/7959049
关键词：
CaliforniaChildhood AsthmaClinicalComputer Retrieval of Information on Scientific Projects DatabaseDevelopmentEventFundingGrantImmuneInstitutionLungMacaca mulattaModelingPrimatesPulmonary EosinophiliaResearchResearch PersonnelResourcesSourceSymptomsUnited States National Institutes of Healthallergic airway diseasepostnatal

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中文摘要
翻译
这个子项目是许多研究子项目中利用 资源由NIH/NCRR资助的中心拨款提供。子项目和 调查员(PI)可能从NIH的另一个来源获得了主要资金， 并因此可以在其他清晰的条目中表示。列出的机构是 该中心不一定是调查人员的机构。 该项目的主要目标是利用过敏性呼吸道疾病的恒河猴模型，确定引发儿童哮喘临床症状发展的早期肺部机制。我们假设肺嗜酸性粒细胞增多症是在出生后发育过程中引起过敏性呼吸道疾病的关键的第一步。
英文摘要
This subproject is one of many research subprojects utilizing the resources provided by a Center grant funded by NIH/NCRR. The subproject and investigator (PI) may have received primary funding from another NIH source, and thus could be represented in other CRISP entries. The institution listed is for the Center, which is not necessarily the institution for the investigator. The primary objective of this project is to determine the early pulmonary mechanisms that initiate development of clinical symptoms in childhood asthma, using a rhesus monkey model of allergic airways disease. We have hypothesized that pulmonary eosinophilia is a critical first step in the initiation of allergic airways disease during postnatal development.
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The Role of Copper in Cerebral Amyloid Angiopathy
  • 批准号：
    9919005
  • 项目类别：
  • 资助金额：
    $38.38万
  • 财政年份：
    2017
  • 负责人：
    LISA M MILLER
  • 依托单位：
    UNIVERSITY OF RHODE ISLAND
The Role of Copper in Cerebral Amyloid Angiopathy
The Role of Copper in Cerebral Amyloid Angiopathy
ROLE OF EPITHELIUM IN AIRWAY IMMUNITY
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